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GWAS Study

Fetal hemoglobin in sickle cell anemia: genome-wide association studies suggest a regulatory region in the 5' olfactory receptor gene cluster.

Solovieff N, Milton JN, Hartley SW et al.

20018918 PubMed ID
GWAS Study Type
1672 Participants
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Chapter I

Publication Details

Comprehensive information about this research publication

Authors

SN
Solovieff N
MJ
Milton JN
HS
Hartley SW
SR
Sherva R
SP
Sebastiani P
DD
Dworkis DA
KE
Klings ES
FL
Farrer LA
GM
Garrett ME
AA
Ashley-Koch A
TM
Telen MJ
FS
Fucharoen S
HS
Ha SY
LC
Li CK
CD
Chui DH
BC
Baldwin CT
SM
Steinberg MH
Chapter II

Abstract

Summary of the research findings

In a genome-wide association study of 848 blacks with sickle cell anemia, we identified single nucleotide polymorphisms (SNPs) associated with fetal hemoglobin concentration. The most significant SNPs in a discovery sample were tested in a replication set of 305 blacks with sickle cell anemia and in subjects with hemoglobin E or beta thalassemia trait from Thailand and Hong Kong. A novel region on chromosome 11 containing olfactory receptor genes OR51B5 and OR51B6 was identified by 6 SNPs (lowest P = 4.7E-08) and validated in the replication set. An additional olfactory receptor gene, OR51B2, was identified by a novel SNP set enrichment analysis. Genome-wide association studies also validated a previously identified SNP (rs766432) in BCL11A, a gene known to affect fetal hemoglobin levels (P = 2.6E-21) and in Thailand and Hong Kong subjects. Elements within the olfactory receptor gene cluster might play a regulatory role in gamma-globin gene expression.

848 Black cases

Chapter III

Study Statistics

Key metrics and study information

1672
Total Participants
GWAS
Study Type
Yes
Replicated
305 Black cases, 406 Chinese ancestry cases, 113 Thai ancestry cases
Replication Participants
South East Asian, East Asian, African unspecified
Ancestry
Thailand, China, Hong Kong SAR
Recruitment Country
Chapter IV

Analysis

Comprehensive review of health and genetic findings

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