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GWAS Study

Common variants at 7p21 are associated with frontotemporal lobar degeneration with TDP-43 inclusions.

Van Deerlin VM, Sleiman PM, Martinez-Lage M et al.

20154673 PubMed ID
GWAS Study Type
3666 Participants
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Chapter I

Publication Details

Comprehensive information about this research publication

Authors

VD
Van Deerlin VM
SP
Sleiman PM
MM
Martinez-Lage M
CA
Chen-Plotkin A
WL
Wang LS
GN
Graff-Radford NR
DD
Dickson DW
RR
Rademakers R
BB
Boeve BF
GM
Grossman M
AS
Arnold SE
MD
Mann DM
PS
Pickering-Brown SM
SH
Seelaar H
HP
Heutink P
VS
van Swieten JC
MJ
Murrell JR
GB
Ghetti B
SS
Spina S
GJ
Grafman J
HJ
Hodges J
SM
Spillantini MG
GS
Gilman S
LA
Lieberman AP
KJ
Kaye JA
WR
Woltjer RL
BE
Bigio EH
MM
Mesulam M
AS
Al-Sarraj S
TC
Troakes C
RR
Rosenberg RN
WC
White CL
FI
Ferrer I
LA
Lladó A
NM
Neumann M
KH
Kretzschmar HA
HC
Hulette CM
WK
Welsh-Bohmer KA
MB
Miller BL
AA
Alzualde A
LD
Lopez de Munain A
MA
McKee AC
GM
Gearing M
LA
Levey AI
LJ
Lah JJ
HJ
Hardy J
RJ
Rohrer JD
LT
Lashley T
MI
Mackenzie IR
FH
Feldman HH
HR
Hamilton RL
DS
Dekosky ST
VD
van der Zee J
KS
Kumar-Singh S
VB
Van Broeckhoven C
MR
Mayeux R
VJ
Vonsattel JP
TJ
Troncoso JC
KJ
Kril JJ
KJ
Kwok JB
HG
Halliday GM
BT
Bird TD
IP
Ince PG
SP
Shaw PJ
CN
Cairns NJ
MJ
Morris JC
MC
McLean CA
DC
DeCarli C
EW
Ellis WG
FS
Freeman SH
FM
Frosch MP
GJ
Growdon JH
PD
Perl DP
SM
Sano M
BD
Bennett DA
SJ
Schneider JA
BT
Beach TG
RE
Reiman EM
WB
Woodruff BK
CJ
Cummings J
VH
Vinters HV
MC
Miller CA
CH
Chui HC
AI
Alafuzoff I
HP
Hartikainen P
SD
Seilhean D
GD
Galasko D
ME
Masliah E
CC
Cotman CW
TM
Tuñón MT
MM
Martínez MC
MD
Munoz DG
CS
Carroll SL
MD
Marson D
RP
Riederer PF
BN
Bogdanovic N
SG
Schellenberg GD
HH
Hakonarson H
TJ
Trojanowski JQ
LV
Lee VM
Chapter II

Abstract

Summary of the research findings

Frontotemporal lobar degeneration (FTLD) is the second most common cause of presenile dementia. The predominant neuropathology is FTLD with TAR DNA-binding protein (TDP-43) inclusions (FTLD-TDP). FTLD-TDP is frequently familial, resulting from mutations in GRN (which encodes progranulin). We assembled an international collaboration to identify susceptibility loci for FTLD-TDP through a genome-wide association study of 515 individuals with FTLD-TDP. We found that FTLD-TDP associates with multiple SNPs mapping to a single linkage disequilibrium block on 7p21 that contains TMEM106B. Three SNPs retained genome-wide significance following Bonferroni correction (top SNP rs1990622, P = 1.08 x 10(-11); odds ratio, minor allele (C) 0.61, 95% CI 0.53-0.71). The association replicated in 89 FTLD-TDP cases (rs1990622; P = 2 x 10(-4)). TMEM106B variants may confer risk of FTLD-TDP by increasing TMEM106B expression. TMEM106B variants also contribute to genetic risk for FTLD-TDP in individuals with mutations in GRN. Our data implicate variants in TMEM106B as a strong risk factor for FTLD-TDP, suggesting an underlying pathogenic mechanism.

515 European ancestry cases, 2,509 European ancestry controls

Chapter III

Study Statistics

Key metrics and study information

3666
Total Participants
GWAS
Study Type
Yes
Replicated
89 European ancestry cases, 553 European ancestry controls
Replication Participants
European
Ancestry
U.S., Australia, Netherlands, Canada, Belgium, U.K., Spain, Finland, Sweden, Germany, France
Recruitment Country
Chapter IV

Analysis

Comprehensive review of health and genetic findings

Important Disclaimer: This review has been performed semi-automatically and is provided for informational purposes only. While we strive for accuracy, this analysis may contain errors, omissions, or misinterpretations of the original research. DNA Genics disclaims all liability for any inaccuracies, errors, or consequences arising from the use of this information. Users should independently verify all information and consult original research publications before making any decisions based on this content. This analysis is not intended as a substitute for professional scientific review or medical advice.

Analysis In Progress

Our analysis of this publication is currently being prepared. Please check back soon for comprehensive insights into the health and genetic findings discussed in this research.