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GWAS Study

CUBN is a gene locus for albuminuria.

Böger CA, Chen MH, Tin A et al.

21355061 PubMed ID
GWAS Study Type
62857 Participants
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Chapter I

Publication Details

Comprehensive information about this research publication

Authors

BC
Böger CA
CM
Chen MH
TA
Tin A
OM
Olden M
KA
Köttgen A
DB
de Boer IH
FC
Fuchsberger C
OC
O'Seaghdha CM
PC
Pattaro C
TA
Teumer A
LC
Liu CT
GN
Glazer NL
LM
Li M
OJ
O'Connell JR
TT
Tanaka T
PC
Peralta CA
KZ
Kutalik Z
LJ
Luan J
ZJ
Zhao JH
HS
Hwang SJ
AE
Akylbekova E
KH
Kramer H
VD
van der Harst P
SA
Smith AV
LK
Lohman K
DA
de Andrade M
HC
Hayward C
KB
Kollerits B
TA
Tönjes A
AT
Aspelund T
IE
Ingelsson E
EG
Eiriksdottir G
LL
Launer LJ
HT
Harris TB
SA
Shuldiner AR
MB
Mitchell BD
AD
Arking DE
FN
Franceschini N
BE
Boerwinkle E
EJ
Egan J
HD
Hernandez D
RM
Reilly M
TR
Townsend RR
LT
Lumley T
SD
Siscovick DS
PB
Psaty BM
KB
Kestenbaum B
HT
Haritunians T
BS
Bergmann S
VP
Vollenweider P
WG
Waeber G
MV
Mooser V
WD
Waterworth D
JA
Johnson AD
FJ
Florez JC
MJ
Meigs JB
LX
Lu X
TS
Turner ST
AE
Atkinson EJ
LT
Leak TS
AK
Aasarød K
SF
Skorpen F
SA
Syvänen AC
IT
Illig T
BJ
Baumert J
KW
Koenig W
KB
Krämer BK
DO
Devuyst O
MJ
Mychaleckyj JC
MC
Minelli C
BS
Bakker SJ
KL
Kedenko L
PB
Paulweber B
CS
Coassin S
EK
Endlich K
KH
Kroemer HK
BR
Biffar R
SS
Stracke S
VH
Völzke H
SM
Stumvoll M
MR
Mägi R
CH
Campbell H
VV
Vitart V
HN
Hastie ND
GV
Gudnason V
KS
Kardia SL
LY
Liu Y
PO
Polasek O
CG
Curhan G
KF
Kronenberg F
PI
Prokopenko I
RI
Rudan I
AJ
Arnlöv J
HS
Hallan S
NG
Navis G
PA
Parsa A
FL
Ferrucci L
CJ
Coresh J
SM
Shlipak MG
BS
Bull SB
PN
Paterson NJ
WH
Wichmann HE
WN
Wareham NJ
LR
Loos RJ
RJ
Rotter JI
PP
Pramstaller PP
CL
Cupples LA
BJ
Beckmann JS
YQ
Yang Q
HI
Heid IM
RR
Rettig R
DA
Dreisbach AW
BM
Bochud M
FC
Fox CS
KW
Kao WH
Chapter II

Abstract

Summary of the research findings

Identification of genetic risk factors for albuminuria may alter strategies for early prevention of CKD progression, particularly among patients with diabetes. Little is known about the influence of common genetic variants on albuminuria in both general and diabetic populations. We performed a meta-analysis of data from 63,153 individuals of European ancestry with genotype information from genome-wide association studies (CKDGen Consortium) and from a large candidate gene study (CARe Consortium) to identify susceptibility loci for the quantitative trait urinary albumin-to-creatinine ratio (UACR) and the clinical diagnosis microalbuminuria. We identified an association between a missense variant (I2984V) in the CUBN gene, which encodes cubilin, and both UACR (P = 1.1 × 10(-11)) and microalbuminuria (P = 0.001). We observed similar associations among 6981 African Americans in the CARe Consortium. The associations between this variant and both UACR and microalbuminuria were significant in individuals of European ancestry regardless of diabetes status. Finally, this variant associated with a 41% increased risk for the development of persistent microalbuminuria during 20 years of follow-up among 1304 participants with type 1 diabetes in the prospective DCCT/EDIC Study. In summary, we identified a missense CUBN variant that associates with levels of albuminuria in both the general population and in individuals with diabetes.

31,580 European ancestry individuals

Chapter III

Study Statistics

Key metrics and study information

62857
Total Participants
GWAS
Study Type
Yes
Replicated
31,277 European ancestry individuals
Replication Participants
European
Ancestry
U.S., Iceland, Germany, Croatia, Italy, U.K., Switzerland
Recruitment Country
Chapter IV

Analysis

Comprehensive review of health and genetic findings

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Analysis In Progress

Our analysis of this publication is currently being prepared. Please check back soon for comprehensive insights into the health and genetic findings discussed in this research.