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GWAS Study

Three new single nucleotide polymorphisms identified by a genome-wide association study in Korean patients with vitiligo.

Cheong KA, Kim NH, Noh M et al.

23678272 PubMed ID
GWAS Study Type
588 Participants
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Chapter I

Publication Details

Comprehensive information about this research publication

Authors

CK
Cheong KA
KN
Kim NH
NM
Noh M
LA
Lee AY
Chapter II

Abstract

Summary of the research findings

Genetic susceptibility is involved in the pathogenesis of vitiligo. Association studies with a whole genome-based approach instead of a single or a few candidate genes may be useful for discovering new susceptible genes. Although the etiology of non-segmental and segmental types is different, the association between gene polymorphisms and vitiligo has been reported, without defining types or in non-segmental type. Whole genome-based single nucleotide polymorphisms (SNPs) were examined in patients with non-segmental and segmental types of vitiligo using the Affymetrix GeneChip 500K mapping array, and 10 functional classes of significant SNPs were selected. Genotyping and data analysis of selected 10 SNPs was performed using real-time PCR. Genotype and allele frequencies were significantly different between both types of vitiligo and three of the target SNPs, DNAH5 (rs2277046), STRN3 (rs2273171), and KIAA1005 (rs3213758). A stronger association was suggested between the mutation in KIAA1005 (rs3213758) and the segmental type compared to the non-segmental type of vitiligo. DNAH5 (rs2277046), STRN3 (rs2273171), and KIAA1005 (rs3213758) may be new vitiligo-related SNPs in Korean patients, either non-segmental or segmental type.

20 Korean ancestry cases, 192 Korean ancestry controls

Chapter III

Study Statistics

Key metrics and study information

588
Total Participants
GWAS
Study Type
Yes
Replicated
184 Korean ancestry cases, 192 Korean ancestry controls
Replication Participants
East Asian
Ancestry
Republic of Korea
Recruitment Country
Chapter IV

Analysis

Comprehensive review of health and genetic findings

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