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GWAS Study

Variation at 3p24.1 and 6q23.3 influences the risk of Hodgkin's lymphoma.

Frampton M, da Silva Filho MI, Broderick P et al.

24149102 PubMed ID
GWAS Study Type
11759 Participants
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Chapter I

Publication Details

Comprehensive information about this research publication

Authors

FM
Frampton M
DS
da Silva Filho MI
BP
Broderick P
TH
Thomsen H
FA
Försti A
VJ
Vijayakrishnan J
CR
Cooke R
EV
Enciso-Mora V
HP
Hoffmann P
NM
Nöthen MM
LA
Lloyd A
HA
Holroyd A
EL
Eisele L
JK
Jöckel KH
PS
Ponader S
VS
von Strandmann EP
LT
Lightfoot T
RE
Roman E
LA
Lake A
MD
Montgomery D
JR
Jarrett RF
SA
Swerdlow AJ
EA
Engert A
HK
Hemminki K
HR
Houlston RS
Chapter II

Abstract

Summary of the research findings

In addition to HLA, recent genome-wide association studies (GWASs) of Hodgkin's lymphoma (HL) have identified susceptibility loci for HL at 2p16.1, 8q24.21 and 10p14. In this study, we perform a GWAS meta-analysis with published GWAS (totalling 1,465 cases and 6,417 controls of European background), and follow-up the most significant association signals in 2,024 cases and 1,853 controls. A combined analysis identifies new HL susceptibility loci mapping to 3p24.1 (rs3806624; P=1.14 × 10(-12), odds ratio (OR)=1.26) and 6q23.3 (rs7745098; P=3.42 × 10(-9), OR=1.21). rs3806624 localizes 5' to the EOMES (eomesodermin) gene within a p53 response element affecting p53 binding. rs7745098 maps intergenic to HBS1L and MYB, a region previously associated with haematopoiesis. These findings provide further insight into the genetic and biological basis of inherited susceptibility to HL.

1,465 European ancestry cases, 6,417 European ancestry controls

Chapter III

Study Statistics

Key metrics and study information

11759
Total Participants
GWAS
Study Type
Yes
Replicated
1,071 European ancestry cases, 953 cases, 1,853 controls
Replication Participants
European
Ancestry
U.K., Germany
Recruitment Country
Chapter IV

Analysis

Comprehensive review of health and genetic findings

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