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GWAS Study

Genome-wide association study of L-arginine and dimethylarginines reveals novel metabolic pathway for symmetric dimethylarginine.

Lüneburg N, Lieb W, Zeller T et al.

25245031 PubMed ID
GWAS Study Type
7898 Participants
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Chapter I

Publication Details

Comprehensive information about this research publication

Authors

LN
Lüneburg N
LW
Lieb W
ZT
Zeller T
CM
Chen MH
MR
Maas R
CA
Carter AM
XV
Xanthakis V
GN
Glazer NL
SE
Schwedhelm E
SS
Seshadri S
IM
Ikram MA
LW
Longstreth WT
FM
Fornage M
KI
König IR
LC
Loley C
OF
Ojeda FM
SA
Schillert A
WT
Wang TJ
SH
Sticht H
KA
Kittel A
KJ
König J
BE
Benjamin EJ
SL
Sullivan LM
BI
Bernges I
AM
Anderssohn M
ZA
Ziegler A
GC
Gieger C
IT
Illig T
MC
Meisinger C
WH
Wichmann HE
WP
Wild PS
SH
Schunkert H
PB
Psaty BM
WK
Wiggins KL
HS
Heckbert SR
SN
Smith N
LK
Lackner K
LK
Lunetta KL
BS
Blankenberg S
EJ
Erdmann J
MT
Munzel T
GP
Grant PJ
VR
Vasan RS
BR
Böger RH
Chapter II

Abstract

Summary of the research findings

Dimethylarginines (DMA) interfere with nitric oxide formation by inhibiting nitric oxide synthase (asymmetrical DMA [ADMA]) and l-arginine uptake into the cell (ADMA and symmetrical DMA [SDMA]). In prospective clinical studies, ADMA has been characterized as a cardiovascular risk marker, whereas SDMA is a novel marker for renal function and associated with all-cause mortality after ischemic stroke. The aim of the current study was to characterize the environmental and genetic contributions to interindividual variability of these biomarkers.

3,747 European ancestry individuals, 2,992 individuals,

Chapter III

Study Statistics

Key metrics and study information

7898
Total Participants
GWAS
Study Type
Yes
Replicated
1,159 European ancestry individuals
Replication Participants
European
Ancestry
Germany, U.S.
Recruitment Country
Chapter IV

Analysis

Comprehensive review of health and genetic findings

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Analysis In Progress

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