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GWAS Study

Large-scale genomic analyses link reproductive aging to hypothalamic signaling, breast cancer susceptibility and BRCA1-mediated DNA repair.

Day FR, Ruth KS, Thompson DJ et al.

26414677 PubMed ID
GWAS Study Type
69626 Participants
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Chapter I

Publication Details

Comprehensive information about this research publication

Authors

DF
Day FR
RK
Ruth KS
TD
Thompson DJ
LK
Lunetta KL
PN
Pervjakova N
CD
Chasman DI
SL
Stolk L
FH
Finucane HK
SP
Sulem P
BB
Bulik-Sullivan B
ET
Esko T
JA
Johnson AD
EC
Elks CE
FN
Franceschini N
HC
He C
AE
Altmaier E
BJ
Brody JA
FL
Franke LL
HJ
Huffman JE
KM
Keller MF
MP
McArdle PF
NT
Nutile T
PE
Porcu E
RA
Robino A
RL
Rose LM
SU
Schick UM
SJ
Smith JA
TA
Teumer A
TM
Traglia M
VD
Vuckovic D
YJ
Yao J
ZW
Zhao W
AE
Albrecht E
AN
Amin N
CT
Corre T
HJ
Hottenga JJ
MM
Mangino M
SA
Smith AV
TT
Tanaka T
AG
Abecasis G
AI
Andrulis IL
AH
Anton-Culver H
AA
Antoniou AC
AV
Arndt V
AA
Arnold AM
BC
Barbieri C
BM
Beckmann MW
BA
Beeghly-Fadiel A
BJ
Benitez J
BL
Bernstein L
BS
Bielinski SJ
BC
Blomqvist C
BE
Boerwinkle E
BN
Bogdanova NV
BS
Bojesen SE
BM
Bolla MK
BA
Borresen-Dale AL
BT
Boutin TS
BH
Brauch H
BH
Brenner H
BT
Brüning T
BB
Burwinkel B
CA
Campbell A
CH
Campbell H
CS
Chanock SJ
CJ
Chapman JR
CY
Chen YI
CG
Chenevix-Trench G
CF
Couch FJ
CA
Coviello AD
CA
Cox A
CK
Czene K
DH
Darabi H
DV
De Vivo I
DE
Demerath EW
DJ
Dennis J
DP
Devilee P
DT
Dörk T
DI
Dos-Santos-Silva I
DA
Dunning AM
EJ
Eicher JD
FP
Fasching PA
FJ
Faul JD
FJ
Figueroa J
FD
Flesch-Janys D
GI
Gandin I
GM
Garcia ME
GM
García-Closas M
GG
Giles GG
GG
Girotto GG
GM
Goldberg MS
GA
González-Neira A
GM
Goodarzi MO
GM
Grove ML
GD
Gudbjartsson DF
GP
Guénel P
GX
Guo X
HC
Haiman CA
HP
Hall P
HU
Hamann U
HB
Henderson BE
HL
Hocking LJ
HA
Hofman A
HG
Homuth G
HM
Hooning MJ
HJ
Hopper JL
HF
Hu FB
HJ
Huang J
HK
Humphreys K
HD
Hunter DJ
JA
Jakubowska A
JS
Jones SE
KM
Kabisch M
KD
Karasik D
KJ
Knight JA
KI
Kolcic I
KC
Kooperberg C
KV
Kosma VM
KJ
Kriebel J
KV
Kristensen V
LD
Lambrechts D
LC
Langenberg C
LJ
Li J
LX
Li X
LS
Lindström S
LY
Liu Y
LJ
Luan J
LJ
Lubinski J
MR
Mägi R
MA
Mannermaa A
MJ
Manz J
MS
Margolin S
MJ
Marten J
MN
Martin NG
MC
Masciullo C
MA
Meindl A
MK
Michailidou K
ME
Mihailov E
ML
Milani L
MR
Milne RL
MM
Müller-Nurasyid M
NM
Nalls M
NB
Neale BM
NH
Nevanlinna H
NP
Neven P
NA
Newman AB
NB
Nordestgaard BG
OJ
Olson JE
PS
Padmanabhan S
PP
Peterlongo P
PU
Peters U
PA
Petersmann A
PJ
Peto J
PP
Pharoah PDP
PN
Pirastu NN
PA
Pirie A
PG
Pistis G
PO
Polasek O
PD
Porteous D
PB
Psaty BM
PK
Pylkäs K
RP
Radice P
RL
Raffel LJ
RF
Rivadeneira F
RI
Rudan I
RA
Rudolph A
RD
Ruggiero D
SC
Sala CF
SS
Sanna S
SE
Sawyer EJ
SD
Schlessinger D
SM
Schmidt MK
SF
Schmidt F
SR
Schmutzler RK
SM
Schoemaker MJ
SR
Scott RA
SC
Seynaeve CM
SJ
Simard J
SR
Sorice R
SM
Southey MC
SD
Stöckl D
SK
Strauch K
SA
Swerdlow A
TK
Taylor KD
TU
Thorsteinsdottir U
TA
Toland AE
TI
Tomlinson I
TT
Truong T
TL
Tryggvadottir L
TS
Turner ST
VD
Vozzi D
WQ
Wang Q
WM
Wellons M
WG
Willemsen G
WJ
Wilson JF
WR
Winqvist R
WB
Wolffenbuttel BBHR
WA
Wright AF
YD
Yannoukakos D
ZT
Zemunik T
ZW
Zheng W
ZM
Zygmunt M
BS
Bergmann S
BD
Boomsma DI
BJ
Buring JE
FL
Ferrucci L
MG
Montgomery GW
GV
Gudnason V
ST
Spector TD
VD
van Duijn CM
AB
Alizadeh BZ
CM
Ciullo M
CL
Crisponi L
ED
Easton DF
GP
Gasparini PP
GC
Gieger C
HT
Harris TB
HC
Hayward C
KS
Kardia SLR
KP
Kraft P
MB
McKnight B
MA
Metspalu A
MA
Morrison AC
RA
Reiner AP
RP
Ridker PM
RJ
Rotter JI
TD
Toniolo D
UA
Uitterlinden AG
US
Ulivi S
VH
Völzke H
WN
Wareham NJ
WD
Weir DR
YL
Yerges-Armstrong LM
PA
Price AL
SK
Stefansson K
VJ
Visser JA
OK
Ong KK
CJ
Chang-Claude J
MJ
Murabito JM
PJ
Perry JRB
MA
Murray A
Chapter II

Abstract

Summary of the research findings

Menopause timing has a substantial impact on infertility and risk of disease, including breast cancer, but the underlying mechanisms are poorly understood. We report a dual strategy in ∼70,000 women to identify common and low-frequency protein-coding variation associated with age at natural menopause (ANM). We identified 44 regions with common variants, including two regions harboring additional rare missense alleles of large effect. We found enrichment of signals in or near genes involved in delayed puberty, highlighting the first molecular links between the onset and end of reproductive lifespan. Pathway analyses identified major association with DNA damage response (DDR) genes, including the first common coding variant in BRCA1 associated with any complex trait. Mendelian randomization analyses supported a causal effect of later ANM on breast cancer risk (∼6% increase in risk per year; P = 3 × 10(-14)), likely mediated by prolonged sex hormone exposure rather than DDR mechanisms.

up to 69,626 European ancestry women

Chapter III

Study Statistics

Key metrics and study information

69626
Total Participants
GWAS
Study Type
Yes
Replicated
up to 46,638 European ancestry women
Replication Participants
European
Ancestry
Chapter IV

Analysis

Comprehensive review of health and genetic findings

Important Disclaimer: This review has been performed semi-automatically and is provided for informational purposes only. While we strive for accuracy, this analysis may contain errors, omissions, or misinterpretations of the original research. DNA Genics disclaims all liability for any inaccuracies, errors, or consequences arising from the use of this information. Users should independently verify all information and consult original research publications before making any decisions based on this content. This analysis is not intended as a substitute for professional scientific review or medical advice.

Analysis In Progress

Our analysis of this publication is currently being prepared. Please check back soon for comprehensive insights into the health and genetic findings discussed in this research.