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GWAS Study

GWAS identifies population-specific new regulatory variants in FUT6 associated with plasma B12 concentrations in Indians.

Nongmaithem SS, Joglekar CV, Krishnaveni GV et al.

28334792 PubMed ID
GWAS Study Type
4419 Participants
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Chapter I

Publication Details

Comprehensive information about this research publication

Authors

NS
Nongmaithem SS
JC
Joglekar CV
KG
Krishnaveni GV
SS
Sahariah SA
AM
Ahmad M
RS
Ramachandran S
GM
Gandhi M
CH
Chopra H
PA
Pandit A
PR
Potdar RD
HD
H D Fall C
YC
Yajnik CS
CG
Chandak GR
Chapter II

Abstract

Summary of the research findings

Vitamin B12 is an important cofactor in one-carbon metabolism whose dysregulation is associated with various clinical conditions. Indians have a high prevalence of B12 deficiency but little is known about the genetic determinants of circulating B12 concentrations in Indians. We performed a genome-wide association study in 1001 healthy participants in the Pune Maternal Nutrition Study (PMNS), replication studies in 3418 individuals from other Indian cohorts and by meta-analysis identified new variants, rs3760775 (P = 1.2 × 10-23) and rs78060698 (P = 8.3 × 10-17) in FUT6 to be associated with circulating B12 concentrations. Although in-silico analysis replicated both variants in Europeans, differences in the effect allele frequency, effect size and the linkage disequilibrium structure of credible set variants with the reported variants suggest population-specific characteristics in this region. We replicated previously reported variants rs602662, rs601338 in FUT2, rs3760776, rs708686 in FUT6, rs34324219 in TCN1 (all P < 5 × 10-8), rs1131603 in TCN2 (P = 3.4 × 10-5), rs12780845 in CUBN (P = 3.0 × 10-3) and rs2270655 in MMAA (P = 2.0 × 10-3). Circulating B12 concentrations in the PMNS and Parthenon study showed a significant decline with increasing age (P < 0.001), however, the genetic contribution to B12 concentrations remained constant. Luciferase reporter and electrophoretic-mobility shift assay for the FUT6 variant rs78060698 using HepG2 cell line demonstrated strong allele-specific promoter and enhancer activity and differential binding of HNF4α, a key regulator of expression of various fucosyltransferases. Hence, the rs78060698 variant, through regulation of fucosylation may control intestinal host-microbial interaction which could influence B12 concentrations. Our results suggest that in addition to established genetic variants, population-specific variants are important in determining plasma B12 concentrations.

1,001 Indo-European ancestry adults

Chapter III

Study Statistics

Key metrics and study information

4419
Total Participants
GWAS
Study Type
Yes
Replicated
1,713 Indo-European ancestry adults, 690 Indo-European ancestry children, 481 Dravidian ancestry adults, 534 Dravidian ancestry children
Replication Participants
South Asian
Ancestry
India
Recruitment Country
Chapter IV

Analysis

Comprehensive review of health and genetic findings

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