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GWAS Study

A genetic variant near GATA3 implicated in inherited susceptibility and etiology of benign prostatic hyperplasia (BPH) and lower urinary tract symptoms (LUTS).

Na R, Helfand BT, Chen H et al.

28656603 PubMed ID
GWAS Study Type
5079 Participants
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Chapter I

Publication Details

Comprehensive information about this research publication

Authors

NR
Na R
HB
Helfand BT
CH
Chen H
CC
Conran CA
CS
Crawford SE
HS
Hayward SW
TT
Tammela TLJ
HJ
Hoffman-Bolton J
ZS
Zheng SL
WP
Walsh PC
SJ
Schleutker J
PE
Platz EA
IW
Isaacs WB
XJ
Xu J
Chapter II

Abstract

Summary of the research findings

Benign prostatic hyperplasia (BPH) and associated lower urinary tract symptoms (LUTS) are common conditions. Little is known about their etiologies except that studies have suggested a substantial heritable component. Our objective is to provide a comprehensive, genome-wide evaluation of inherited risks and possible mechanisms of etiology in BPH.

1,419 European ancestry cases, 498 cases, 1,684 European ancestry controls, 518 controls

Chapter III

Study Statistics

Key metrics and study information

5079
Total Participants
GWAS
Study Type
Yes
Replicated
485 Finnish ancestry cases, 475 Finnish ancestry controls
Replication Participants
European
Ancestry
Finland, U.S.
Recruitment Country
Chapter IV

Analysis

Comprehensive review of health and genetic findings

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Analysis In Progress

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