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GWAS Study

Exome-wide association study of plasma lipids in >300,000 individuals.

Liu DJ, Peloso GM, Yu H et al.

29083408 PubMed ID
GWAS Study Type
584092 Participants
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Chapter I

Publication Details

Comprehensive information about this research publication

Authors

LD
Liu DJ
PG
Peloso GM
YH
Yu H
BA
Butterworth AS
WX
Wang X
MA
Mahajan A
SD
Saleheen D
EC
Emdin C
AD
Alam D
AA
Alves AC
AP
Amouyel P
DA
Di Angelantonio E
AD
Arveiler D
AT
Assimes TL
AP
Auer PL
BU
Baber U
BC
Ballantyne CM
BL
Bang LE
BM
Benn M
BJ
Bis JC
BM
Boehnke M
BE
Boerwinkle E
BJ
Bork-Jensen J
BE
Bottinger EP
BI
Brandslund I
BM
Brown M
BF
Busonero F
CM
Caulfield MJ
CJ
Chambers JC
CD
Chasman DI
CY
Chen YE
CY
Chen YI
CR
Chowdhury R
CC
Christensen C
CA
Chu AY
CJ
Connell JM
CF
Cucca F
CL
Cupples LA
DS
Damrauer SM
DG
Davies G
DI
Deary IJ
DG
Dedoussis G
DJ
Denny JC
DA
Dominiczak A
DM
Dubé MP
ET
Ebeling T
EG
Eiriksdottir G
ET
Esko T
FA
Farmaki AE
FM
Feitosa MF
FM
Ferrario M
FJ
Ferrieres J
FI
Ford I
FM
Fornage M
FP
Franks PW
FT
Frayling TM
FR
Frikke-Schmidt R
FL
Fritsche LG
FP
Frossard P
FV
Fuster V
GS
Ganesh SK
GW
Gao W
GM
Garcia ME
GC
Gieger C
GF
Giulianini F
GM
Goodarzi MO
GH
Grallert H
GN
Grarup N
GL
Groop L
GM
Grove ML
GV
Gudnason V
HT
Hansen T
HT
Harris TB
HC
Hayward C
HJ
Hirschhorn JN
HO
Holmen OL
HJ
Huffman J
HY
Huo Y
HK
Hveem K
JS
Jabeen S
JA
Jackson AU
JJ
Jakobsdottir J
JM
Jarvelin MR
JG
Jensen GB
JM
Jørgensen ME
JJ
Jukema JW
JJ
Justesen JM
KP
Kamstrup PR
KS
Kanoni S
KF
Karpe F
KF
Kee F
KA
Khera AV
KD
Klarin D
KH
Koistinen HA
KJ
Kooner JS
KC
Kooperberg C
KK
Kuulasmaa K
KJ
Kuusisto J
LM
Laakso M
LT
Lakka T
LC
Langenberg C
LA
Langsted A
LL
Launer LJ
LT
Lauritzen T
LD
Liewald DCM
LL
Lin LA
LA
Linneberg A
LR
Loos RJF
LY
Lu Y
LX
Lu X
MR
Mägi R
MA
Malarstig A
MA
Manichaikul A
MA
Manning AK
MP
Mäntyselkä P
ME
Marouli E
MN
Masca NGD
MA
Maschio A
MJ
Meigs JB
MO
Melander O
MA
Metspalu A
MA
Morris AP
MA
Morrison AC
MA
Mulas A
MM
Müller-Nurasyid M
MP
Munroe PB
NM
Neville MJ
NJ
Nielsen JB
NS
Nielsen SF
NB
Nordestgaard BG
OJ
Ordovas JM
MR
Mehran R
OC
O'Donnell CJ
OM
Orho-Melander M
MC
Molony CM
MP
Muntendam P
PS
Padmanabhan S
PC
Palmer CNA
PD
Pasko D
PA
Patel AP
PO
Pedersen O
PM
Perola M
PA
Peters A
PC
Pisinger C
PG
Pistis G
PO
Polasek O
PN
Poulter N
PB
Psaty BM
RD
Rader DJ
RA
Rasheed A
RR
Rauramaa R
RD
Reilly DF
RA
Reiner AP
RF
Renström F
RS
Rich SS
RP
Ridker PM
RJ
Rioux JD
RN
Robertson NR
RD
Roden DM
RJ
Rotter JI
RI
Rudan I
SV
Salomaa V
SN
Samani NJ
SS
Sanna S
SN
Sattar N
SE
Schmidt EM
SR
Scott RA
SP
Sever P
SR
Sevilla RS
SC
Shaffer CM
SX
Sim X
SS
Sivapalaratnam S
SK
Small KS
SA
Smith AV
SB
Smith BH
SS
Somayajula S
SL
Southam L
ST
Spector TD
SE
Speliotes EK
SJ
Starr JM
SK
Stirrups KE
SN
Stitziel N
SK
Strauch K
SH
Stringham HM
SP
Surendran P
TH
Tada H
TA
Tall AR
TH
Tang H
TJ
Tardif JC
TK
Taylor KD
TS
Trompet S
TP
Tsao PS
TJ
Tuomilehto J
TA
Tybjaerg-Hansen A
VZ
van Zuydam NR
VA
Varbo A
VT
Varga TV
VJ
Virtamo J
WM
Waldenberger M
WN
Wang N
WN
Wareham NJ
WH
Warren HR
WP
Weeke PE
WJ
Weinstock J
WJ
Wessel J
WJ
Wilson JG
WP
Wilson PWF
XM
Xu M
YH
Yaghootkar H
YR
Young R
ZE
Zeggini E
ZH
Zhang H
ZN
Zheng NS
ZW
Zhang W
ZY
Zhang Y
ZW
Zhou W
ZY
Zhou Y
ZM
Zoledziewska M
HJ
Howson JMM
DJ
Danesh J
MM
McCarthy MI
CC
Cowan CA
AG
Abecasis G
DP
Deloukas P
MK
Musunuru K
WC
Willer CJ
KS
Kathiresan S
Chapter II

Abstract

Summary of the research findings

We screened variants on an exome-focused genotyping array in >300,000 participants (replication in >280,000 participants) and identified 444 independent variants in 250 loci significantly associated with total cholesterol (TC), high-density-lipoprotein cholesterol (HDL-C), low-density-lipoprotein cholesterol (LDL-C), and/or triglycerides (TG). At two loci (JAK2 and A1CF), experimental analysis in mice showed lipid changes consistent with the human data. We also found that: (i) beta-thalassemia trait carriers displayed lower TC and were protected from coronary artery disease (CAD); (ii) excluding the CETP locus, there was not a predictable relationship between plasma HDL-C and risk for age-related macular degeneration; (iii) only some mechanisms of lowering LDL-C appeared to increase risk for type 2 diabetes (T2D); and (iv) TG-lowering alleles involved in hepatic production of TG-rich lipoproteins (TM6SF2 and PNPLA3) tracked with higher liver fat, higher risk for T2D, and lower risk for CAD, whereas TG-lowering alleles involved in peripheral lipolysis (LPL and ANGPTL4) had no effect on liver fat but decreased risks for both T2D and CAD.

up to 237,050 European ancestry individuals, up to 16,935 African ancestry individuals, up to 30,468 South Asian ancestry individuals, up to 8,287 East Asian ancestry individuals, 5,084 Hispanic individuals

Chapter III

Study Statistics

Key metrics and study information

584092
Total Participants
GWAS
Study Type
Yes
Replicated
up to 286,268 European and unknown ancestry individuals
Replication Participants
East Asian, South Asian, European, NR, Hispanic or Latin American, African unspecified, European
Ancestry
U.S., China, Bangladesh, Pakistan, U.K., Norway, Germany, Netherlands, Denmark, Estonia, Finland, Iceland, Republic of Ireland, Sweden, Croatia, Greece, Italy
Recruitment Country
Chapter IV

Analysis

Comprehensive review of health and genetic findings

Important Disclaimer: This review has been performed semi-automatically and is provided for informational purposes only. While we strive for accuracy, this analysis may contain errors, omissions, or misinterpretations of the original research. DNA Genics disclaims all liability for any inaccuracies, errors, or consequences arising from the use of this information. Users should independently verify all information and consult original research publications before making any decisions based on this content. This analysis is not intended as a substitute for professional scientific review or medical advice.

Analysis In Progress

Our analysis of this publication is currently being prepared. Please check back soon for comprehensive insights into the health and genetic findings discussed in this research.