Menu
GWAS Study

Cross-ancestry genome-wide association analysis of corneal thickness strengthens link between complex and Mendelian eye diseases.

Iglesias AI, Mishra A, Vitart V et al.

29760442 PubMed ID
GWAS Study Type
25910 Participants
Scroll to explore
Chapter I

Publication Details

Comprehensive information about this research publication

Authors

IA
Iglesias AI
MA
Mishra A
VV
Vitart V
BY
Bykhovskaya Y
HR
Höhn R
SH
Springelkamp H
CG
Cuellar-Partida G
GP
Gharahkhani P
BJ
Bailey JNC
WC
Willoughby CE
LX
Li X
YS
Yazar S
NA
Nag A
KA
Khawaja AP
PO
Polašek O
SD
Siscovick D
MP
Mitchell P
TY
Tham YC
HJ
Haines JL
KL
Kearns LS
HC
Hayward C
SY
Shi Y
VL
van Leeuwen EM
TK
Taylor KD
BP
Bonnemaijer P
RJ
Rotter JI
MN
Martin NG
ZT
Zeller T
MR
Mills RA
SE
Souzeau E
SS
Staffieri SE
JJ
Jonas JB
SI
Schmidtmann I
BT
Boutin T
KJ
Kang JH
LS
Lucas SEM
WT
Wong TY
BM
Beutel ME
WJ
Wilson JF
UA
Uitterlinden AG
VE
Vithana EN
FP
Foster PJ
HP
Hysi PG
HA
Hewitt AW
KC
Khor CC
PL
Pasquale LR
MG
Montgomery GW
KC
Klaver CCW
AT
Aung T
PN
Pfeiffer N
MD
Mackey DA
HC
Hammond CJ
CC
Cheng CY
CJ
Craig JE
RY
Rabinowitz YS
WJ
Wiggs JL
BK
Burdon KP
VD
van Duijn CM
MS
MacGregor S
Chapter II

Abstract

Summary of the research findings

Central corneal thickness (CCT) is a highly heritable trait associated with complex eye diseases such as keratoconus and glaucoma. We perform a genome-wide association meta-analysis of CCT and identify 19 novel regions. In addition to adding support for known connective tissue-related pathways, pathway analyses uncover previously unreported gene sets. Remarkably, >20% of the CCT-loci are near or within Mendelian disorder genes. These included FBN1, ADAMTS2 and TGFB2 which associate with connective tissue disorders (Marfan, Ehlers-Danlos and Loeys-Dietz syndromes), and the LUM-DCN-KERA gene complex involved in myopia, corneal dystrophies and cornea plana. Using index CCT-increasing variants, we find a significant inverse correlation in effect sizes between CCT and keratoconus (r = -0.62, P = 5.30 × 10-5) but not between CCT and primary open-angle glaucoma (r = -0.17, P = 0.2). Our findings provide evidence for shared genetic influences between CCT and keratoconus, and implicate candidate genes acting in collagen and extracellular matrix regulation.

17,803 European ancestry individuals, 8,107 Asian ancestry individuals

Chapter III

Study Statistics

Key metrics and study information

25910
Total Participants
GWAS
Study Type
No
Replicated
East Asian, South Asian, South East Asian, European
Ancestry
Chapter IV

Analysis

Comprehensive review of health and genetic findings

Important Disclaimer: This review has been performed semi-automatically and is provided for informational purposes only. While we strive for accuracy, this analysis may contain errors, omissions, or misinterpretations of the original research. DNA Genics disclaims all liability for any inaccuracies, errors, or consequences arising from the use of this information. Users should independently verify all information and consult original research publications before making any decisions based on this content. This analysis is not intended as a substitute for professional scientific review or medical advice.

Analysis In Progress

Our analysis of this publication is currently being prepared. Please check back soon for comprehensive insights into the health and genetic findings discussed in this research.