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GWAS Study

Bivariate genome-wide association analyses of the broad depression phenotype combined with major depressive disorder, bipolar disorder or schizophrenia reveal eight novel genetic loci for depression.

Amare AT, Vaez A, Hsu YH et al.

30626913 PubMed ID
GWAS Study Type
699951 Participants
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Chapter I

Publication Details

Comprehensive information about this research publication

Authors

AA
Amare AT
VA
Vaez A
HY
Hsu YH
DN
Direk N
KZ
Kamali Z
HD
Howard DM
MA
McIntosh AM
TH
Tiemeier H
BU
Bültmann U
SH
Snieder H
HC
Hartman CA
Chapter II

Abstract

Summary of the research findings

Although a genetic basis of depression has been well established in twin studies, identification of genome-wide significant loci has been difficult. We hypothesized that bivariate analyses of findings from a meta-analysis of genome-wide association studies (meta-GWASs) of the broad depression phenotype with those from meta-GWASs of self-reported and recurrent major depressive disorder (MDD), bipolar disorder and schizophrenia would enhance statistical power to identify novel genetic loci for depression. LD score regression analyses were first used to estimate the genetic correlations of broad depression with self-reported MDD, recurrent MDD, bipolar disorder and schizophrenia. Then, we performed four bivariate GWAS analyses. The genetic correlations (rg ± SE) of broad depression with self-reported MDD, recurrent MDD, bipolar disorder and schizophrenia were 0.79 ± 0.07, 0.24 ± 0.08, 0.53 ± 0.09 and 0.57 ± 0.05, respectively. From a total of 20 independent genome-wide significant loci, 13 loci replicated of which 8 were novel for depression. These were MUC21 for the broad depression phenotype with self-reported MDD and ZNF804A, MIR3143, PSORS1C2, STK19, SPATA31D1, RTN1 and TCF4 for the broad depression phenotype with schizophrenia. Post-GWAS functional analyses of these loci revealed their potential biological involvement in psychiatric disorders. Our results emphasize the genetic similarities among different psychiatric disorders and indicate that cross-disorder analyses may be the best way forward to accelerate gene finding for depression, or psychiatric disorders in general.

9,240 European ancestry broad depression cases, 9,519 European ancestry broad depression controls, 51,258 European ancestry individuals, 75,607 European ancestry self-reported major depressive disorder cases, 231,747 European ancestry self-reported major depressive disorder controls

Chapter III

Study Statistics

Key metrics and study information

699951
Total Participants
GWAS
Study Type
Yes
Replicated
322,580 individuals
Replication Participants
European, East Asian
Ancestry
Chapter IV

Analysis

Comprehensive review of health and genetic findings

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