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GWAS Study

Drug-induced injury due to flucloxacillin: relevance of multiple HLA alleles.

Nicoletti P, Aithal GP, Chamberlain TC et al.

30661239 PubMed ID
GWAS Study Type
7032 Participants
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Chapter I

Publication Details

Comprehensive information about this research publication

Authors

NP
Nicoletti P
AG
Aithal GP
CT
Chamberlain TC
CS
Coulthard S
AM
Alshabeeb M
GJ
Grove JI
AR
Andrade RJ
BE
Bjornsson E
DJ
Dillon JF
HP
Hallberg P
LM
Lucena MI
MD
Maitland-van der Zee AH
MJ
Martin JH
MM
Molokhia M
PM
Pirmohamed M
WM
Wadelius M
SY
Shen Y
NM
Nelson MR
DA
Daly AK
Chapter II

Abstract

Summary of the research findings

Some patients prescribed flucloxacillin (~ 0.01%) develop drug-induced liver injury (DILI). HLA-B*57:01 is an established genetic risk factor for flucloxacillin DILI. To consolidate this finding, identify additional genetic factors, and assess relevance of risk factors for flucloxacillin DILI in relation to DILI due to other penicillins, we performed a genomewide association study involving 197 flucloxacillin DILI cases and 6,835 controls. We imputed single-nucleotide polymorphism and human leukocyte antigen (HLA) genotypes. HLA-B*57:01 was the major risk factor (allelic odds ratio (OR) = 36.62; P = 2.67 × 10-97 ). HLA-B*57:03 also showed an association (OR = 79.21; P = 1.2 × 10-6 ). Within the HLA-B protein sequence, imputation showed valine97 , common to HLA-B*57:01 and HLA-B*57:03, had the largest effect (OR = 38.1; P = 9.7 × 10-97 ). We found no HLA-B*57 association with DILI due to other isoxazolyl penicillins (n = 6) or amoxicillin (n = 15) and no significant non-HLA signals for any penicillin-related DILI.

197 European ancestry cases, 6,835 European ancestry controls

Chapter III

Study Statistics

Key metrics and study information

7032
Total Participants
GWAS
Study Type
No
Replicated
European
Ancestry
Australia, Netherlands, Sweden, U.K., U.S., Iceland
Recruitment Country
Chapter IV

Analysis

Comprehensive review of health and genetic findings

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