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GWAS Study

Genome-wide meta-analysis of depression identifies 102 independent variants and highlights the importance of the prefrontal brain regions.

Howard DM, Adams MJ, Clarke TK et al.

30718901 PubMed ID
GWAS Study Type
2113907 Participants
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Chapter I

Publication Details

Comprehensive information about this research publication

Authors

HD
Howard DM
AM
Adams MJ
CT
Clarke TK
HJ
Hafferty JD
GJ
Gibson J
SM
Shirali M
CJ
Coleman JRI
HS
Hagenaars SP
WJ
Ward J
WE
Wigmore EM
AC
Alloza C
SX
Shen X
BM
Barbu MC
XE
Xu EY
WH
Whalley HC
MR
Marioni RE
PD
Porteous DJ
DG
Davies G
DI
Deary IJ
HG
Hemani G
BK
Berger K
TH
Teismann H
RR
Rawal R
AV
Arolt V
BB
Baune BT
DU
Dannlowski U
DK
Domschke K
TC
Tian C
HD
Hinds DA
TM
Trzaskowski M
BE
Byrne EM
RS
Ripke S
SD
Smith DJ
SP
Sullivan PF
WN
Wray NR
BG
Breen G
LC
Lewis CM
MA
McIntosh AM
Chapter II

Abstract

Summary of the research findings

Major depression is a debilitating psychiatric illness that is typically associated with low mood and anhedonia. Depression has a heritable component that has remained difficult to elucidate with current sample sizes due to the polygenic nature of the disorder. To maximize sample size, we meta-analyzed data on 807,553 individuals (246,363 cases and 561,190 controls) from the three largest genome-wide association studies of depression. We identified 102 independent variants, 269 genes, and 15 genesets associated with depression, including both genes and gene pathways associated with synaptic structure and neurotransmission. An enrichment analysis provided further evidence of the importance of prefrontal brain regions. In an independent replication sample of 1,306,354 individuals (414,055 cases and 892,299 controls), 87 of the 102 associated variants were significant after multiple testing correction. These findings advance our understanding of the complex genetic architecture of depression and provide several future avenues for understanding etiology and developing new treatment approaches.

118,811 European ancestry cases, 327,427 European ancestry controls, 127,552 cases, 233,763 controls

Chapter III

Study Statistics

Key metrics and study information

2113907
Total Participants
GWAS
Study Type
Yes
Replicated
414,055 European ancestry cases, 892,299 European ancestry controls
Replication Participants
European
Ancestry
Chapter IV

Analysis

Comprehensive review of health and genetic findings

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Analysis In Progress

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