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GWAS Study

Non-del(5q) myelodysplastic syndromes-associated loci detected by SNP-array genome-wide association meta-analysis.

McGraw KL, Cheng CH, Chen YA et al.

31738830 PubMed ID
GWAS Study Type
6512 Participants
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Chapter I

Publication Details

Comprehensive information about this research publication

Authors

MK
McGraw KL
CC
Cheng CH
CY
Chen YA
HH
Hou HA
NB
Nilsson B
GG
Genovese G
CT
Cluzeau T
PA
Pellagatti A
PB
Przychodzen BP
MM
Mallo M
AL
Arenillas L
MA
Mohamedali A
AL
Adès L
SD
Sallman DA
PE
Padron E
SL
Sokol L
MC
Moreilhon C
RS
Raynaud S
TH
Tien HF
BJ
Boultwood J
EB
Ebert BL
SF
Sole F
FP
Fenaux P
MG
Mufti GJ
MJ
Maciejewski JP
KP
Kanetsky PA
LA
List AF
Chapter II

Abstract

Summary of the research findings

Myelodysplastic syndromes (MDS) are hematopoietic stem cell malignancies. Known predisposing factors to adult MDS include rare germline mutations, cytotoxic therapy, age-related clonal hematopoiesis, and autoimmune or chronic inflammatory disorders. To date, no published studies characterizing MDS-associated germline susceptibility polymorphisms exist. We performed a genome-wide association study of 2 sample sets (555 MDS cases vs 2964 control subjects; 352 MDS cases vs 2640 control subjects) in non-del(5q) MDS cases of European genomic ancestry. Meta-analysis identified 8 MDS-associated loci at 1q31.1 (PLA2G4A), 3p14.1 (FAM19A4), 5q21.3 (EFNA5), 6p21.33, 10q23.1 (GRID1), 12q24.32, 15q26.1, and 20q13.12 (EYA2) that approached genome-wide significance. Gene expression for 5 loci that mapped within or near genes was significantly upregulated in MDS bone marrow cells compared with those of control subjects (P < .01). Higher PLA2G4A expression and lower EYA2 expression were associated with poorer overall survival (P = .039 and P = .037, respectively). Higher PLA2G4A expression is associated with mutations in NRAS (P < .001), RUNX1 (P = .012), ASXL1 (P = .007), and EZH2 (P = .038), all of which are known to contribute to MDS development. EYA2 expression was an independently favorable risk factor irrespective of age, sex, and Revised International Scoring System score (relative risk, 0.67; P = .048). Notably, these genes have regulatory roles in innate immunity, a critical driver of MDS pathogenesis. EYA2 overexpression induced innate immune activation, whereas EYA2 inhibition restored colony-forming potential in primary MDS cells indicative of hematopoietic restoration and possible clinical relevance. In conclusion, among 8 suggestive MDS-associated loci, 5 map to genes upregulated in MDS with functional roles in innate immunity and potential biological relevance to MDS.

907 European ancestry cases, 5,605 European ancestry controls

Chapter III

Study Statistics

Key metrics and study information

6512
Total Participants
GWAS
Study Type
No
Replicated
European
Ancestry
France, U.S., U.K., Spain
Recruitment Country
Chapter IV

Analysis

Comprehensive review of health and genetic findings

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