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GWAS Study

Genome-Wide Association Study of Ocular Sarcoidosis Confirms HLA Associations and Implicates Barrier Function and Autoimmunity in African Americans.

Garman L, Pezant N, Pastori A et al.

32141793 PubMed ID
GWAS Study Type
1811 Participants
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Chapter I

Publication Details

Comprehensive information about this research publication

Authors

GL
Garman L
PN
Pezant N
PA
Pastori A
SK
Savoy KA
LC
Li C
LA
Levin AM
IM
Iannuzzi MC
RB
Rybicki BA
AI
Adrianto I
MC
Montgomery CG
Chapter II

Abstract

Summary of the research findings

Purpose: Identify genes associated with ocular sarcoidosis (OS).Methods: We genotyped 1.1 million genetic variants to identify significant OS associations, defined as those that achieved p < 5 × 10-8 in a genome-wide comparison of OS cases to healthy controls in our European- or African-American cohorts (EA, AA). Potential functional roles of all associated variants were assessed.Results: Eight significant non-HLA variants were found in AA OS cases compared to healthy controls and confirmed as at least suggestive when comparing OS to non-OS cases. Seven of these were within MAGI1 and include transcription factor binding sites and expression quantitative trait loci. Our EA cohort, while showing similar effect sizes at variants within MAGI1, had no significant variants. Association analysis of HLA-DRB1 alleles confirmed association to OS in EA to *04:01.Conclusion: Our results support organ-specific genetic risk in OS in a compelling candidate, MAGI1, known to be associated with barrier function and autoimmunity.

260 African American cases, 1,551 African American controls

Chapter III

Study Statistics

Key metrics and study information

1811
Total Participants
GWAS
Study Type
No
Replicated
African American or Afro-Caribbean, European
Ancestry
U.S.
Recruitment Country
Chapter IV

Analysis

Comprehensive review of health and genetic findings

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