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GWAS Study

Shared genetic background between children and adults with attention deficit/hyperactivity disorder.

Rovira P, Demontis D, Sánchez-Mora C et al.

32279069 PubMed ID
GWAS Study Type
49560 Participants
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Chapter I

Publication Details

Comprehensive information about this research publication

Authors

RP
Rovira P
DD
Demontis D
SC
Sánchez-Mora C
ZT
Zayats T
KM
Klein M
MN
Mota NR
WH
Weber H
GI
Garcia-Martínez I
PM
Pagerols M
VL
Vilar-Ribó L
AL
Arribas L
RV
Richarte V
CM
Corrales M
FC
Fadeuilhe C
BR
Bosch R
MG
Martin GE
AP
Almos P
DA
Doyle AE
GE
Grevet EH
GO
Grimm O
HA
Halmøy A
HM
Hoogman M
HM
Hutz M
JC
Jacob CP
KS
Kittel-Schneider S
KP
Knappskog PM
LA
Lundervold AJ
RO
Rivero O
RD
Rovaris DL
SA
Salatino-Oliveira A
DS
da Silva BS
SE
Svirin E
SE
Sprooten E
ST
Strekalova T
AA
Arias-Vasquez A
SE
Sonuga-Barke EJS
AP
Asherson P
BC
Bau CHD
BJ
Buitelaar JK
CB
Cormand B
FS
Faraone SV
HJ
Haavik J
JS
Johansson SE
KJ
Kuntsi J
LH
Larsson H
LK
Lesch KP
RA
Reif A
RL
Rohde LA
CM
Casas M
BA
Børglum AD
FB
Franke B
RJ
Ramos-Quiroga JA
SA
Soler Artigas M
RM
Ribasés M
Chapter II

Abstract

Summary of the research findings

Attention deficit/hyperactivity disorder (ADHD) is a common neurodevelopmental disorder characterized by age-inappropriate symptoms of inattention, impulsivity, and hyperactivity that persist into adulthood in the majority of the diagnosed children. Despite several risk factors during childhood predicting the persistence of ADHD symptoms into adulthood, the genetic architecture underlying the trajectory of ADHD over time is still unclear. We set out to study the contribution of common genetic variants to the risk for ADHD across the lifespan by conducting meta-analyses of genome-wide association studies on persistent ADHD in adults and ADHD in childhood separately and jointly, and by comparing the genetic background between them in a total sample of 17,149 cases and 32,411 controls. Our results show nine new independent loci and support a shared contribution of common genetic variants to ADHD in children and adults. No subgroup heterogeneity was observed among children, while this group consists of future remitting and persistent individuals. We report similar patterns of genetic correlation of ADHD with other ADHD-related datasets and different traits and disorders among adults, children, and when combining both groups. These findings confirm that persistent ADHD in adults is a neurodevelopmental disorder and extend the existing hypothesis of a shared genetic architecture underlying ADHD and different traits to a lifespan perspective.

17,149 European ancestry cases, 32,411 European ancestry controls

Chapter III

Study Statistics

Key metrics and study information

49560
Total Participants
GWAS
Study Type
No
Replicated
European
Ancestry
Norway, Spain, Denmark, Germany, Netherlands, Brazil
Recruitment Country
Chapter IV

Analysis

Comprehensive review of health and genetic findings

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