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GWAS Study

The Genetics of Circulating Resistin Level, A Biomarker for Cardiovascular Diseases, Is Informed by Mendelian Randomization and the Unique Characteristics of African Genomes.

Meeks KAC, Doumatey AP, Bentley AR et al.

32876488 PubMed ID
GWAS Study Type
5621 Participants
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Chapter I

Publication Details

Comprehensive information about this research publication

Authors

MK
Meeks KAC
DA
Doumatey AP
BA
Bentley AR
GM
Gouveia MH
CG
Chen G
ZJ
Zhou J
LL
Lei L
AA
Adeyemo AA
RC
Rotimi CN
Chapter II

Abstract

Summary of the research findings

Resistin, a protein linked with inflammation and cardiometabolic diseases, is one of few proteins for which genome-wide association studies consistently report variants within and near the coding gene (RETN). Here, we took advantage of the reduced linkage disequilibrium in African populations to infer genetic causality for circulating resistin levels by performing genome-wide association studies, whole-exome analysis, fine mapping, Mendelian randomization, and transcriptomic data analyses.

3,754 Sub-Saharan African ancestry individuals, 1,867 African American individuals

Chapter III

Study Statistics

Key metrics and study information

5621
Total Participants
GWAS
Study Type
No
Replicated
Sub-Saharan African, African American or Afro-Caribbean
Ancestry
Ghana, Nigeria, U.S.
Recruitment Country
Chapter IV

Analysis

Comprehensive review of health and genetic findings

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