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GWAS Study

Genomic and drug target evaluation of 90 cardiovascular proteins in 30,931 individuals.

Folkersen L, Gustafsson S, Wang Q et al.

33067605 PubMed ID
GWAS Study Type
30931 Participants
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Chapter I

Publication Details

Comprehensive information about this research publication

Authors

FL
Folkersen L
GS
Gustafsson S
WQ
Wang Q
HD
Hansen DH
Hedman ÅK
SA
Schork A
PK
Page K
ZD
Zhernakova DV
WY
Wu Y
PJ
Peters J
EN
Eriksson N
BS
Bergen SE
BT
Boutin TS
BA
Bretherick AD
ES
Enroth S
KA
Kalnapenkis A
GJ
Gådin JR
SB
Suur BE
CY
Chen Y
ML
Matic L
GJ
Gale JD
LJ
Lee J
ZW
Zhang W
QA
Quazi A
AM
Ala-Korpela M
CS
Choi SH
CA
Claringbould A
DJ
Danesh J
DS
Davey Smith G
DM
de Masi F
ES
Elmståhl S
EG
Engström G
FE
Fauman E
FC
Fernandez C
FL
Franke L
FP
Franks PW
GV
Giedraitis V
HC
Haley C
HA
Hamsten A
IA
Ingason A
Johansson Å
JP
Joshi PK
LL
Lind L
LC
Lindgren CM
LS
Lubitz S
PT
Palmer T
ME
Macdonald-Dunlop E
MM
Magnusson M
MO
Melander O
MK
Michaelsson K
MA
Morris AP
MR
Mägi R
NM
Nagle MW
NP
Nilsson PM
NJ
Nilsson J
OM
Orho-Melander M
PO
Polasek O
PB
Prins B
PE
Pålsson E
QT
Qi T
SM
Sjögren M
SJ
Sundström J
SP
Surendran P
VU
Võsa U
WT
Werge T
WR
Wernersson R
WH
Westra HJ
YJ
Yang J
ZA
Zhernakova A
ÄJ
Ärnlöv J
FJ
Fu J
SJ
Smith JG
ET
Esko T
HC
Hayward C
GU
Gyllensten U
LM
Landen M
SA
Siegbahn A
WJ
Wilson JF
WL
Wallentin L
BA
Butterworth AS
HM
Holmes MV
IE
Ingelsson E
MA
Mälarstig A
Chapter II

Abstract

Summary of the research findings

Circulating proteins are vital in human health and disease and are frequently used as biomarkers for clinical decision-making or as targets for pharmacological intervention. Here, we map and replicate protein quantitative trait loci (pQTL) for 90 cardiovascular proteins in over 30,000 individuals, resulting in 451 pQTLs for 85 proteins. For each protein, we further perform pathway mapping to obtain trans-pQTL gene and regulatory designations. We substantiate these regulatory findings with orthogonal evidence for trans-pQTLs using mouse knockdown experiments (ABCA1 and TRIB1) and clinical trial results (chemokine receptors CCR2 and CCR5), with consistent regulation. Finally, we evaluate known drug targets, and suggest new target candidates or repositioning opportunities using Mendelian randomization. This identifies 11 proteins with causal evidence of involvement in human disease that have not previously been targeted, including EGF, IL-16, PAPPA, SPON1, F3, ADM, CASP-8, CHI3L1, CXCL16, GDF15 and MMP-12. Taken together, these findings demonstrate the utility of large-scale mapping of the genetics of the proteome and provide a resource for future precision studies of circulating proteins in human health.

21,758 European ancestry individuals

Chapter III

Study Statistics

Key metrics and study information

30931
Total Participants
GWAS
Study Type
Yes
Replicated
9,173 individuals
Replication Participants
European
Ancestry
Sweden
Recruitment Country
Chapter IV

Analysis

Comprehensive review of health and genetic findings

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Analysis In Progress

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