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GWAS Study

The FAM171A2 gene is a key regulator of progranulin expression and modifies the risk of multiple neurodegenerative diseases.

Xu W, Han SD, Zhang C et al.

33087363 PubMed ID
GWAS Study Type
1362 Participants
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Chapter I

Publication Details

Comprehensive information about this research publication

Authors

XW
Xu W
HS
Han SD
ZC
Zhang C
LJ
Li JQ
WY
Wang YJ
TC
Tan CC
LH
Li HQ
DQ
Dong Q
MC
Mei C
TL
Tan L
YJ
Yu JT
Chapter II

Abstract

Summary of the research findings

Progranulin (PGRN) is a secreted pleiotropic glycoprotein associated with the development of common neurodegenerative diseases. Understanding the pathophysiological role of PGRN may help uncover biological underpinnings. We performed a genome-wide association study to determine the genetic regulators of cerebrospinal fluid (CSF) PGRN levels. Common variants in region of FAM171A2 were associated with lower CSF PGRN levels (rs708384, P = 3.95 × 10-12). This was replicated in another independent cohort. The rs708384 was associated with increased risk of Alzheimer's disease, Parkinson's disease, and frontotemporal dementia and could modify the expression of the FAM171A2 gene. FAM171A2 was considerably expressed in the vascular endothelium and microglia, which are rich in PGRN. The in vitro study further confirmed that the rs708384 mutation up-regulated the expression of FAM171A2, which caused a decrease in the PGRN level. Collectively, genetic, molecular, and bioinformatic findings suggested that FAM171A2 is a key player in regulating PGRN production.

432 European ancestry individuals

Chapter III

Study Statistics

Key metrics and study information

1362
Total Participants
GWAS
Study Type
Yes
Replicated
930 Han Chinese ancestry individuals
Replication Participants
European, East Asian
Ancestry
China
Recruitment Country
Chapter IV

Analysis

Comprehensive review of health and genetic findings

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