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GWAS Study

A functional genomics approach in Tanzanian population identifies distinct genetic regulators of cytokine production compared to European population.

Boahen CK, Temba GS, Kullaya VI et al.

35167808 PubMed ID
GWAS Study Type
271 Participants
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Chapter I

Publication Details

Comprehensive information about this research publication

Authors

BC
Boahen CK
TG
Temba GS
KV
Kullaya VI
MV
Matzaraki V
JL
Joosten LAB
KG
Kibiki G
MB
Mmbaga BT
VD
van der Ven A
DM
de Mast Q
NM
Netea MG
KV
Kumar V
Chapter II

Abstract

Summary of the research findings

Humans exhibit remarkable interindividual and interpopulation immune response variability upon microbial challenges. Cytokines play a vital role in regulating inflammation and immune responses, but dysregulation of cytokine responses has been implicated in different disease states. Host genetic factors were previously shown to significantly impact cytokine response heterogeneity mainly in European-based studies, but it is unclear whether these findings are transferable to non-European individuals. Here, we aimed to identify genetic variants modulating cytokine responses in healthy adults of East African ancestry from Tanzania. We leveraged both cytokine and genetic data and performed genome-wide cytokine quantitative trait loci (cQTLs) mapping. The results were compared with another cohort of healthy adults of Western European ancestry via direct overlap and functional enrichment analyses. We also performed meta-analyses to identify cQTLs with congruent effect direction in both populations. In the Tanzanians, cQTL mapping identified 80 independent suggestive loci and one genome-wide significant locus (TBC1D22A) at chromosome 22; SNP rs12169244 was associated with IL-1b release after Salmonella enteritidis stimulation. Remarkably, the identified cQTLs varied significantly when compared to the European cohort, and there was a very limited percentage of overlap (1.6% to 1.9%). We further observed ancestry-specific pathways regulating induced cytokine responses, and there was significant enrichment of the interferon pathway specifically in the Tanzanians. Furthermore, contrary to the Europeans, genetic variants in the TLR10-TLR1-TLR6 locus showed no effect on cytokine response. Our data reveal both ancestry-specific effects of genetic variants and pathways on cytokine response heterogeneity, hence arguing for the importance of initiatives to include diverse populations into genomics research.

271 Tanzanian ancestry individuals

Chapter III

Study Statistics

Key metrics and study information

271
Total Participants
GWAS
Study Type
No
Replicated
Sub-Saharan African, European
Ancestry
United Republic of Tanzania
Recruitment Country
Chapter IV

Analysis

Comprehensive review of health and genetic findings

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Analysis In Progress

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