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GWAS Study

Genome-wide analyses identify <i>SCN5A</i> as a susceptibility locus for premature atrial contraction frequency.

Thériault S, Imboden M, Biggs ML et al.

36267918 PubMed ID
GWAS Study Type
4831 Participants
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Chapter I

Publication Details

Comprehensive information about this research publication

Authors

TS
Thériault S
IM
Imboden M
BM
Biggs ML
AT
Austin TR
AS
Aeschbacher S
SE
Schaffner E
BJ
Brody JA
BT
Bartz TM
RM
Risch M
GK
Grossmann K
LH
Lin HJ
SE
Soliman EZ
PW
Post WS
RL
Risch L
KJ
Krieger JE
PA
Pereira AC
HS
Heckbert SR
SN
Sotoodehnia N
PN
Probst-Hensch NM
CD
Conen D
Chapter II

Abstract

Summary of the research findings

Premature atrial contractions (PACs) are frequently observed on electrocardiograms and are associated with increased risks of atrial fibrillation (AF), stroke, and mortality. In this study, we aimed to identify genetic susceptibility loci for PAC frequency. We performed a genome-wide association study meta-analysis with PAC frequency obtained from ambulatory cardiac monitoring in 4,831 individuals of European ancestry. We identified a genome-wide significant locus at the SCN5A gene. The lead variant, rs7373862, located in an intron of SCN5A, was associated with an increase of 0.12 [95% CI 0.08-0.16] standard deviations of the normalized PAC frequency per risk allele. Among genetic variants previously associated with AF, there was a significant enrichment in concordance of effect for PAC frequency (n = 73/106, p = 5.1 × 10-5). However, several AF risk loci, including PITX2, were not associated with PAC frequency. These findings suggest the existence of both shared and distinct genetic mechanisms for PAC frequency and AF.

4,831 European ancestry individuals

Chapter III

Study Statistics

Key metrics and study information

4831
Total Participants
GWAS
Study Type
No
Replicated
European
Ancestry
U.S., Liechtenstein, Brazil, Switzerland
Recruitment Country
Chapter IV

Analysis

Comprehensive review of health and genetic findings

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