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GWAS Study

A Genome-Wide Association Study Identified Novel Genetic Susceptibility Loci for Oral Cancer in Taiwan.

Bau DT, Liu TY, Tsai CW et al.

36769103 PubMed ID
GWAS Study Type
46101 Participants
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Chapter I

Publication Details

Comprehensive information about this research publication

Authors

BD
Bau DT
LT
Liu TY
TC
Tsai CW
CW
Chang WS
GJ
Gu J
YJ
Yang JS
SL
Shih LC
TF
Tsai FJ
Chapter II

Abstract

Summary of the research findings

Taiwan has the highest incidence rate of oral cancer in the world. Although oral cancer is mostly an environmentally induced cancer, genetic factors also play an important role in its etiology. Genome-wide association studies (GWAS) have identified nine susceptibility regions for oral cancers in populations of European descent. In this study, we performed the first GWAS of oral cancer in Taiwan with 1529 cases and 44,572 controls. We confirmed two previously reported loci on the 6p21.33 (HLA-B) and 6p21.32 (HLA-DQ gene cluster) loci, highlighting the importance of the human leukocyte antigen and, hence, the immunologic mechanisms in oral carcinogenesis. The TERT-CLMPT1L locus on 5p15.33, the 4q23 ADH1B locus, and the LAMC3 locus on 9q34.12 were also consistent in the Taiwanese. We found two new independent loci on 6p21.32, rs401775 in SKIV2L gene and rs9267798 in TNXB gene. We also found two suggestive novel Taiwanese-specific loci near the TPRS1 gene on 8q23.3 and in the TMED3 gene on 15q25.1. This study identified both common and unique oral cancer susceptibility loci in the Taiwanese as compared to populations of European descent and shed significant light on the etiology of oral cancer in Taiwan.

1,529 Taiwanese ancestry cases, 44,572 Taiwanese ancestry controls

Chapter III

Study Statistics

Key metrics and study information

46101
Total Participants
GWAS
Study Type
No
Replicated
East Asian
Ancestry
Taiwan
Recruitment Country
Chapter IV

Analysis

Comprehensive review of health and genetic findings

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