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GWAS Study

Genetic architecture distinguishes tinnitus from hearing loss.

Clifford RE, Maihofer AX, Chatzinakos C et al.

38242899 PubMed ID
GWAS Study Type
481874 Participants
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Chapter I

Publication Details

Comprehensive information about this research publication

Authors

CR
Clifford RE
MA
Maihofer AX
CC
Chatzinakos C
CJ
Coleman JRI
DN
Daskalakis NP
GM
Gasperi M
HK
Hogan K
ME
Mikita EA
SM
Stein MB
TC
Tcheandjieu C
TF
Telese F
ZY
Zuo Y
RA
Ryan AF
NC
Nievergelt CM
Chapter II

Abstract

Summary of the research findings

Tinnitus is a heritable, highly prevalent auditory disorder treated by multiple medical specialties. Previous GWAS indicated high genetic correlations between tinnitus and hearing loss, with little indication of differentiating signals. We present a GWAS meta-analysis, triple previous sample sizes, and expand to non-European ancestries. GWAS in 596,905 Million Veteran Program subjects identified 39 tinnitus loci, and identified genes related to neuronal synapses and cochlear structural support. Applying state-of-the-art analytic tools, we confirm a large number of shared variants, but also a distinct genetic architecture of tinnitus, with higher polygenicity and large proportion of variants not shared with hearing difficulty. Tissue-expression analysis for tinnitus infers broad enrichment across most brain tissues, in contrast to hearing difficulty. Finally, tinnitus is not only correlated with hearing loss, but also with a spectrum of psychiatric disorders, providing potential new avenues for treatment. This study establishes tinnitus as a distinct disorder separate from hearing difficulties.

481,874 European ancestry individuals

Chapter III

Study Statistics

Key metrics and study information

481874
Total Participants
GWAS
Study Type
No
Replicated
European, Hispanic or Latin American, African unspecified
Ancestry
U.K.
Recruitment Country
Chapter IV

Analysis

Comprehensive review of health and genetic findings

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