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GWAS Study

Genetic drivers of heterogeneity in type 2 diabetes pathophysiology.

Suzuki K, Hatzikotoulas K, Southam L et al.

38374256 PubMed ID
GWAS Study Type
2535601 Participants
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Chapter I

Publication Details

Comprehensive information about this research publication

Authors

SK
Suzuki K
HK
Hatzikotoulas K
SL
Southam L
TH
Taylor HJ
YX
Yin X
LK
Lorenz KM
MR
Mandla R
HA
Huerta-Chagoya A
MG
Melloni GEM
KS
Kanoni S
RN
Rayner NW
BO
Bocher O
AA
Arruda AL
SK
Sonehara K
NS
Namba S
LS
Lee SSK
PM
Preuss MH
PL
Petty LE
SP
Schroeder P
VB
Vanderwerff B
KM
Kals M
BF
Bragg F
LK
Lin K
GX
Guo X
ZW
Zhang W
YJ
Yao J
KY
Kim YJ
GM
Graff M
TF
Takeuchi F
NJ
Nano J
LA
Lamri A
NM
Nakatochi M
MS
Moon S
SR
Scott RA
CJ
Cook JP
LJ
Lee JJ
PI
Pan I
TD
Taliun D
PE
Parra EJ
CJ
Chai JF
BL
Bielak LF
TY
Tabara Y
HY
Hai Y
TG
Thorleifsson G
GN
Grarup N
ST
Sofer T
WM
Wuttke M
SC
Sarnowski C
GC
Gieger C
ND
Nousome D
TS
Trompet S
KS
Kwak SH
LJ
Long J
SM
Sun M
TL
Tong L
CW
Chen WM
NS
Nongmaithem SS
NR
Noordam R
LV
Lim VJY
TC
Tam CHT
JY
Joo YY
CC
Chen CH
RL
Raffield LM
PB
Prins BP
NA
Nicolas A
YL
Yanek LR
CG
Chen G
BJ
Brody JA
KE
Kabagambe E
AP
An P
XA
Xiang AH
CH
Choi HS
CB
Cade BE
TJ
Tan J
BK
Broadaway KA
WA
Williamson A
KZ
Kamali Z
CJ
Cui J
TM
Thangam M
AL
Adair LS
AA
Adeyemo A
AC
Aguilar-Salinas CA
AT
Ahluwalia TS
AS
Anand SS
BA
Bertoni A
BJ
Bork-Jensen J
BI
Brandslund I
BT
Buchanan TA
BC
Burant CF
BA
Butterworth AS
CM
Canouil M
CJ
Chan JCN
CL
Chang LC
CM
Chee ML
CJ
Chen J
CS
Chen SH
CY
Chen YT
CZ
Chen Z
CL
Chuang LM
CM
Cushman M
DJ
Danesh J
DS
Das SK
DS
de Silva HJ
DG
Dedoussis G
DL
Dimitrov L
DA
Doumatey AP
DS
Du S
DQ
Duan Q
EK
Eckardt KU
EL
Emery LS
ED
Evans DS
EM
Evans MK
FK
Fischer K
FJ
Floyd JS
FI
Ford I
FO
Franco OH
FT
Frayling TM
FB
Freedman BI
GP
Genter P
GH
Gerstein HC
GV
Giedraitis V
GC
González-Villalpando C
GM
González-Villalpando ME
GP
Gordon-Larsen P
GM
Gross M
GL
Guare LA
HS
Hackinger S
HL
Hakaste L
HS
Han S
HA
Hattersley AT
HC
Herder C
HM
Horikoshi M
HA
Howard AG
HW
Hsueh W
HM
Huang M
HW
Huang W
HY
Hung YJ
HM
Hwang MY
HC
Hwu CM
IS
Ichihara S
IM
Ikram MA
IM
Ingelsson M
IM
Islam MT
IM
Isono M
JH
Jang HM
JF
Jasmine F
JG
Jiang G
JJ
Jonas JB
JT
Jørgensen T
KF
Kamanu FK
KF
Kandeel FR
KA
Kasturiratne A
KT
Katsuya T
KV
Kaur V
KT
Kawaguchi T
KJ
Keaton JM
KA
Kho AN
KC
Khor CC
KM
Kibriya MG
KD
Kim DH
KF
Kronenberg F
KJ
Kuusisto J
LK
Läll K
LL
Lange LA
LK
Lee KM
LM
Lee MS
LN
Lee NR
LA
Leong A
LL
Li L
LY
Li Y
LR
Li-Gao R
LS
Ligthart S
LC
Lindgren CM
LA
Linneberg A
LC
Liu CT
LJ
Liu J
LA
Locke AE
LT
Louie T
LJ
Luan J
LA
Luk AO
LX
Luo X
LJ
Lv J
LJ
Lynch JA
LV
Lyssenko V
MS
Maeda S
MV
Mamakou V
MS
Mansuri SR
MK
Matsuda K
MT
Meitinger T
MO
Melander O
MA
Metspalu A
MH
Mo H
MA
Morris AD
MF
Moura FA
NJ
Nadler JL
NM
Nalls MA
NU
Nayak U
NI
Ntalla I
OY
Okada Y
OL
Orozco L
PS
Patel SR
PS
Patil S
PP
Pei P
PM
Pereira MA
PA
Peters A
PF
Pirie FJ
PH
Polikowsky HG
PB
Porneala B
PG
Prasad G
RL
Rasmussen-Torvik LJ
RA
Reiner AP
RM
Roden M
RR
Rohde R
RK
Roll K
SC
Sabanayagam C
SK
Sandow K
SA
Sankareswaran A
SN
Sattar N
SS
Schönherr S
SM
Shahriar M
SB
Shen B
SJ
Shi J
SD
Shin DM
SN
Shojima N
SJ
Smith JA
SW
So WY
SA
Stančáková A
SV
Steinthorsdottir V
SA
Stilp AM
SK
Strauch K
TK
Taylor KD
TB
Thorand B
TU
Thorsteinsdottir U
TB
Tomlinson B
TT
Tran TC
TF
Tsai FJ
TJ
Tuomilehto J
TT
Tusie-Luna T
UM
Udler MS
VA
Valladares-Salgado A
VD
van Dam RM
VK
van Klinken JB
VR
Varma R
WN
Wacher-Rodarte N
WE
Wheeler E
WA
Wickremasinghe AR
VD
van Dijk KW
WD
Witte DR
YC
Yajnik CS
YK
Yamamoto K
YK
Yamamoto K
YK
Yoon K
YC
Yu C
YJ
Yuan JM
YS
Yusuf S
ZM
Zawistowski M
ZL
Zhang L
ZW
Zheng W
RL
Raffel LJ
IM
Igase M
IE
Ipp E
RS
Redline S
CY
Cho YS
LL
Lind L
PM
Province MA
FM
Fornage M
HC
Hanis CL
IE
Ingelsson E
ZA
Zonderman AB
PB
Psaty BM
WY
Wang YX
RC
Rotimi CN
BD
Becker DM
MF
Matsuda F
LY
Liu Y
YM
Yokota M
KS
Kardia SLR
PP
Peyser PA
PJ
Pankow JS
EJ
Engert JC
BA
Bonnefond A
FP
Froguel P
WJ
Wilson JG
SW
Sheu WHH
WJ
Wu JY
HM
Hayes MG
MR
Ma RCW
WT
Wong TY
MD
Mook-Kanamori DO
TT
Tuomi T
CG
Chandak GR
CF
Collins FS
BD
Bharadwaj D
PG
Paré G
SM
Sale MM
AH
Ahsan H
MA
Motala AA
SX
Shu XO
PK
Park KS
JJ
Jukema JW
CM
Cruz M
CY
Chen YI
RS
Rich SS
MR
McKean-Cowdin R
GH
Grallert H
CC
Cheng CY
GM
Ghanbari M
TE
Tai ES
DJ
Dupuis J
KN
Kato N
LM
Laakso M
KA
Köttgen A
KW
Koh WP
BD
Bowden DW
PC
Palmer CNA
KJ
Kooner JS
KC
Kooperberg C
LS
Liu S
NK
North KE
SD
Saleheen D
HT
Hansen T
PO
Pedersen O
WN
Wareham NJ
LJ
Lee J
KB
Kim BJ
MI
Millwood IY
WR
Walters RG
SK
Stefansson K
AE
Ahlqvist E
GM
Goodarzi MO
MK
Mohlke KL
LC
Langenberg C
HC
Haiman CA
LR
Loos RJF
FJ
Florez JC
RD
Rader DJ
RM
Ritchie MD
ZS
Zöllner S
MR
Mägi R
MN
Marston NA
RC
Ruff CT
VH
van Heel DA
FS
Finer S
DJ
Denny JC
YT
Yamauchi T
KT
Kadowaki T
CJ
Chambers JC
NM
Ng MCY
SX
Sim X
BJ
Below JE
TP
Tsao PS
CK
Chang KM
MM
McCarthy MI
MJ
Meigs JB
MA
Mahajan A
SC
Spracklen CN
MJ
Mercader JM
BM
Boehnke M
RJ
Rotter JI
VM
Vujkovic M
VB
Voight BF
MA
Morris AP
ZE
Zeggini E
Chapter II

Abstract

Summary of the research findings

Type 2 diabetes (T2D) is a heterogeneous disease that develops through diverse pathophysiological processes1,2 and molecular mechanisms that are often specific to cell type3,4. Here, to characterize the genetic contribution to these processes across ancestry groups, we aggregate genome-wide association study data from 2,535,601 individuals (39.7% not of European ancestry), including 428,452 cases of T2D. We identify 1,289 independent association signals at genome-wide significance (P < 5 × 10-8) that map to 611 loci, of which 145 loci are, to our knowledge, previously unreported. We define eight non-overlapping clusters of T2D signals that are characterized by distinct profiles of cardiometabolic trait associations. These clusters are differentially enriched for cell-type-specific regions of open chromatin, including pancreatic islets, adipocytes, endothelial cells and enteroendocrine cells. We build cluster-specific partitioned polygenic scores5 in a further 279,552 individuals of diverse ancestry, including 30,288 cases of T2D, and test their association with T2D-related vascular outcomes. Cluster-specific partitioned polygenic scores are associated with coronary artery disease, peripheral artery disease and end-stage diabetic nephropathy across ancestry groups, highlighting the importance of obesity-related processes in the development of vascular outcomes. Our findings show the value of integrating multi-ancestry genome-wide association study data with single-cell epigenomics to disentangle the aetiological heterogeneity that drives the development and progression of T2D. This might offer a route to optimize global access to genetically informed diabetes care.

50,251 African American cases, 103,909 African American controls, 88,109 East Asian ancestry cases, 339,395 East Asian ancestry controls, 242,283 European ancestry cases, 1,569,734 European ancestry controls, 29,375 Hispanic cases, 59,368 Hispanic controls, 1,602 South African ancestry cases, 976 South African ancestry controls, 16,832 South Asian ancestry cases, 33,767 South Asian ancestry controls

Chapter III

Study Statistics

Key metrics and study information

2535601
Total Participants
GWAS
Study Type
No
Replicated
African American or Afro-Caribbean, East Asian, European, Hispanic or Latin American, Other, South Asian
Ancestry
U.S., Singapore, Japan, China, Philippines, Taiwan, Republic of Korea, Greece, Sweden, Netherlands, Finland, Denmark, U.K., France, Iceland, Germany, Estonia, Mexico, South Africa, Bangladesh, Pakistan, Sri Lanka, India
Recruitment Country
Chapter IV

Analysis

Comprehensive review of health and genetic findings

Important Disclaimer: This review has been performed semi-automatically and is provided for informational purposes only. While we strive for accuracy, this analysis may contain errors, omissions, or misinterpretations of the original research. DNA Genics disclaims all liability for any inaccuracies, errors, or consequences arising from the use of this information. Users should independently verify all information and consult original research publications before making any decisions based on this content. This analysis is not intended as a substitute for professional scientific review or medical advice.

Analysis In Progress

Our analysis of this publication is currently being prepared. Please check back soon for comprehensive insights into the health and genetic findings discussed in this research.