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GWAS Study

Identifying atopic dermatitis risk loci in 1,094,060 individuals with sub analysis of disease severity and onset.

Pasanen A, Sliz E, Huilaja L et al.

38663478 PubMed ID
GWAS Study Type
1094060 Participants
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Chapter I

Publication Details

Comprehensive information about this research publication

Authors

PA
Pasanen A
SE
Sliz E
HL
Huilaja L
RE
Reimann E
MR
Mägi R
LT
Laisk T
TK
Tasanen K
KJ
Kettunen J
Chapter II

Abstract

Summary of the research findings

Atopic dermatitis (AD) is a common inflammatory skin disease highly attributable to genetic factors. In this study, we report results from a genome-wide meta-analysis of AD in 37,541 cases and 1,056,519 controls with data from the FinnGen project, the Estonian Biobank, the UK Biobank, the EAGLE Consortium, and the BioBank Japan. We detected 77 independent AD-associated loci, of which 10 were, to our knowledge, previously unreported. The associated loci showed enrichment in various immune regulatory processes. We further performed subgroup analyses of mild and severe AD and of early- and late-onset AD, with data from the FinnGen project. Fifty-five of the 79 tested variants in the associated loci showed larger effect estimates for severe than for mild AD as determined through administered treatment. The age of onset, as determined by the first hospital visit with AD diagnosis, was lower in patients with particular AD-risk alleles. Our findings add to the knowledge of the genetic background of AD and may underlie the development of new therapeutic strategies.

35,156 European ancestry cases, 846,868 European ancestry controls, 2,385 Japanese ancestry cases, 209,651 Japanese ancestry controls

Chapter III

Study Statistics

Key metrics and study information

1094060
Total Participants
GWAS
Study Type
No
Replicated
European, East Asian
Ancestry
Finland, U.K., Estonia, Japan
Recruitment Country
Chapter IV

Analysis

Comprehensive review of health and genetic findings

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