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GWAS Study

Genome-wide association study of early-stage non-small cell lung cancer prognosis: a pooled analysis in the International Lung Cancer Consortium.

Dong M, Thakral A, Byrne KS et al.

40746155 PubMed ID
GWAS Study Type
3916 Participants
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Chapter I

Publication Details

Comprehensive information about this research publication

Authors

DM
Dong M
TA
Thakral A
BK
Byrne KS
BY
Bosse Y
ZH
Zhou H
ZY
Zhang Y
AJ
Atkins J
HP
Haycock P
BM
Brown MC
MK
Murison K
TW
Timens W
SD
Sin DD
KJ
Kothari J
GA
Gabriel AAG
ZD
Zaridze D
SM
Savic M
LJ
Lissowska J
ŚB
Świątkowska B
JV
Janout V
HI
Holcatova I
MA
Mukeria A
FG
Fernandez-Tardon G
DM
Davies MPA
TM
Triplette M
SM
Schabath MB
AA
Andrew AS
CC
Chen C
TF
Taylor F
FJ
Field JK
TA
Tardon A
SS
Shete SS
BP
Brennan P
LM
Landi MT
MJ
McKay J
AC
Amos CI
LX
Lin X
CD
Christiani DC
HR
Hung RJ
LG
Liu G
XW
Xu W
Chapter II

Abstract

Summary of the research findings

Lung cancer is the leading cause of cancer mortality. To investigate genetic determinants for prognosis among patients diagnosed with early-stage non-small cell lung cancer (NSCLC), we conducted the first large-scale genome-wide association prognostic study using data from the International Lung Cancer Consortium (ILCCO) through a two-phase analysis. Phase 1 includes the discovery of genome-wide association studies analysis using a multivariable Cox PH model on 3428 NSCLC patients of European ancestry from 10 ILCCO participating studies to identify genetic variants associated with overall survival and validation analysis for genome-wide significant variants (P-value ≤5 × 10-8) using the Cancer Genome Atlas (TCGA). Phase 2 aims to identify causal variants using functional analyses of genome-wide significant and suggestive variants (P-value ≤1 × 10-5), including variant-epigenetic functional annotation (FAVOR), CHIP-seq data, variant-gene expression association, and colocalization analysis. We identified two significant variants; of those, a locus at 9q21.31 (rs117979484) was significant at the genome-wide level (P = 3.67 × 10-8) and validated in TCGA (P = 0.03). Three suggestive variants were found to have a putative epigenetic function: intronic variants rs149281784 (BCL7B gene) and rs148031766 (POM121 gene) both located at 7q11.23 and in moderate linkage disequilibrium with each other; and variant rs2471630 (SRCIN1 gene; 17q12). Specifically, variants rs149281784 and rs148031766 have potential regulatory roles in the transcriptional activation of the BCL7B gene and POM121 gene. Exploratory survival analyses in the squamous cell carcinomas subgroup also identified a significant variant, rs138467404 (GRHL-2 gene; 8q22.3) at a genome-wide level (P = 4.75 × 10-8) and validated by TCGA (P = 0.02). These new findings indicate potential novel pathways associated with early-stage NSCLC prognosis. Future research may validate additional genome-wide suggestive variants as being relevant for lung cancer outcomes.

3,916 European ancestry individuals

Chapter III

Study Statistics

Key metrics and study information

3916
Total Participants
GWAS
Study Type
Yes
Replicated
391 individuals
Replication Participants
European
Ancestry
Canada, Russian Federation, U.S., U.K., Spain
Recruitment Country
Chapter IV

Analysis

Comprehensive review of health and genetic findings

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Analysis In Progress

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