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GWAS Study

An integration of genome-wide association study and gene expression profiling to prioritize the discovery of novel susceptibility Loci for osteoporosis-related traits.

Hsu YH, Zillikens MC, Wilson SG et al.

20548944 PubMed ID
GWAS Study Type
11290 Participants
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Chapter I

Publication Details

Comprehensive information about this research publication

Authors

HY
Hsu YH
ZM
Zillikens MC
WS
Wilson SG
FC
Farber CR
DS
Demissie S
SN
Soranzo N
BE
Bianchi EN
GE
Grundberg E
LL
Liang L
RJ
Richards JB
EK
Estrada K
ZY
Zhou Y
VN
van Nas A
MM
Moffatt MF
ZG
Zhai G
HA
Hofman A
VM
van Meurs JB
PH
Pols HA
PR
Price RI
NO
Nilsson O
PT
Pastinen T
CL
Cupples LA
LA
Lusis AJ
SE
Schadt EE
FS
Ferrari S
UA
Uitterlinden AG
RF
Rivadeneira F
ST
Spector TD
KD
Karasik D
KD
Kiel DP
Chapter II

Abstract

Summary of the research findings

Osteoporosis is a complex disorder and commonly leads to fractures in elderly persons. Genome-wide association studies (GWAS) have become an unbiased approach to identify variations in the genome that potentially affect health. However, the genetic variants identified so far only explain a small proportion of the heritability for complex traits. Due to the modest genetic effect size and inadequate power, true association signals may not be revealed based on a stringent genome-wide significance threshold. Here, we take advantage of SNP and transcript arrays and integrate GWAS and expression signature profiling relevant to the skeletal system in cellular and animal models to prioritize the discovery of novel candidate genes for osteoporosis-related traits, including bone mineral density (BMD) at the lumbar spine (LS) and femoral neck (FN), as well as geometric indices of the hip (femoral neck-shaft angle, NSA; femoral neck length, NL; and narrow-neck width, NW). A two-stage meta-analysis of GWAS from 7,633 Caucasian women and 3,657 men, revealed three novel loci associated with osteoporosis-related traits, including chromosome 1p13.2 (RAP1A, p = 3.6x10(-8)), 2q11.2 (TBC1D8), and 18q11.2 (OSBPL1A), and confirmed a previously reported region near TNFRSF11B/OPG gene. We also prioritized 16 suggestive genome-wide significant candidate genes based on their potential involvement in skeletal metabolism. Among them, 3 candidate genes were associated with BMD in women. Notably, 2 out of these 3 genes (GPR177, p = 2.6x10(-13); SOX6, p = 6.4x10(-10)) associated with BMD in women have been successfully replicated in a large-scale meta-analysis of BMD, but none of the non-prioritized candidates (associated with BMD) did. Our results support the concept of our prioritization strategy. In the absence of direct biological support for identified genes, we highlighted the efficiency of subsequent functional characterization using publicly available expression profiling relevant to the skeletal system in cellular or whole animal models to prioritize candidate genes for further functional validation.

2,038 European ancestry female individuals, 1,531 European ancestry male individuals

Chapter III

Study Statistics

Key metrics and study information

11290
Total Participants
GWAS
Study Type
Yes
Replicated
5,595 European ancestry female individuals, 2,126 European ancestry male individuals
Replication Participants
European
Ancestry
U.K.
Recruitment Country
Chapter IV

Analysis

Comprehensive review of health and genetic findings

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