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GWAS Study

Genome-Wide Association and Replication Study of Hepatotoxicity Induced by Antiretrovirals Alone or with Concomitant Anti-Tuberculosis Drugs.

Petros Z, Lee MT, Takahashi A et al.

28388302 PubMed ID
GWAS Study Type
380 Participants
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Chapter I

Publication Details

Comprehensive information about this research publication

Authors

PZ
Petros Z
LM
Lee MT
TA
Takahashi A
ZY
Zhang Y
YG
Yimer G
HA
Habtewold A
SI
Schuppe-Koistinen I
MT
Mushiroda T
ME
Makonnen E
KM
Kubo M
AE
Aklillu E
Chapter II

Abstract

Summary of the research findings

Drug-induced hepatotoxicity (DIH) is a common adverse event that is associated with both antiretroviral (ARV) and anti-tuberculosis drugs (ATD). Moreover, the genetic variations predisposing ARV- and ARV-ATD-induced liver toxicity in African populations are not well investigated, despite the two diseases being the major global health problems in sub-Saharan Africa. We performed a genome-wide association study (GWAS) and replication study to identify the genetic variants linked to the risk of developing DIH due to ARV drugs alone, and ARV-ATD co-treatment in Ethiopian HIV-positive patients. Treatment-naïve newly diagnosed HIV patients (n = 719) with or without tuberculosis (TB) co-infection were enrolled prospectively and received efavirenz-based ARV therapy with or without rifampicin-based short course ATD, respectively. Whole-genome genotyping was performed by using the Illumina Omni Express Exome Bead Chip genotyping array with 951,117 single nucleotide polymorphisms (SNPs) on a total of 41 cases of DIH, and 452 people without DIH (treatment tolerants). The replication study was carried out for 100 SNPs with the lowest p-values (top SNPs) by using an independent cohort consisting of 18 DIH cases and 208 treatment tolerants. We identified a missense SNP rs199650082 (2756G→A, R919Q, p = 1.4 × 10-6, odds ratio [OR] = 18.2, 95% confidence interval [CI] = 7.1-46.9) in an endoplasmic reticulum to the nucleus signaling-1 (ERN1) gene on chromosome 17 to be associated with DIH in the ARV-only cohort. In the ARV-ATD co-treatment groups, rs4842407, a long intergenic noncoding RNAs (lincRNAs) transcript variant on chromosome 12, was associated with DIH (p = 5.3 × 10-7, OR = 5.4, 95% CI = 2.8-10.3). These genetic variants that are putatively associated with DIH due to ARV drugs alone and ARV-ATD co-treatment establish a foundation for future personalized medicine in people with HIV and TB and call for larger studies in independent populations.

14 Ethiopian cases with HIV, 293 Ethiopian controls with HIV.

Chapter III

Study Statistics

Key metrics and study information

380
Total Participants
GWAS
Study Type
Yes
Replicated
7 Ethiopian cases with HIV, 66 Ethiopian controls with HIV.
Replication Participants
African unspecified
Ancestry
Ethiopia
Recruitment Country
Chapter IV

Analysis

Comprehensive review of health and genetic findings

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