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GWAS Study

Genome-phenome wide association study of broadly defined headache.

Hsu WT, Lee YT, Tan J et al.

37288313 PubMed ID
GWAS Study Type
108855 Participants
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Chapter I

Publication Details

Comprehensive information about this research publication

Authors

HW
Hsu WT
LY
Lee YT
TJ
Tan J
CY
Chang YH
QF
Qian F
LK
Liu KY
HJ
Hsiung JC
YC
Yo CH
TS
Tang SC
JX
Jiang X
LC
Lee CC
Chapter II

Abstract

Summary of the research findings

Until recently, most genetic studies of headache have been conducted on participants with European ancestry. We therefore conducted a large-scale genome-wide association study of self-reported headache in individuals of East Asian ancestry (specifically those who were identified as Han Chinese). In this study, 108 855 participants were enrolled, including 12 026 headache cases from the Taiwan Biobank. For broadly defined headache phenotype, we identified a locus on Chromosome 17, with the lead single-nucleotide polymorphism rs8072917 (odds ratio 1.08, P = 4.49 × 10-8), mapped to two protein-coding genes RNF213 and ENDOV. For severe headache phenotype, we found a strong association on Chromosome 8, with the lead single-nucleotide polymorphism rs13272202 (odds ratio 1.30, P = 1.02 × 10-9), mapped to gene RP11-1101K5.1. We then conducted a conditional analysis and a statistical fine-mapping of the broadly defined headache-associated loci and identified a single credible set of loci with rs8072917 supporting that this lead variant was the true causal variant on RNF213 gene region. RNF213 replicated the result of previous studies and played important roles in the biological mechanism of broadly defined headache. On the basis of the previous results found in the Taiwan Biobank, we conducted phenome-wide association studies for the lead variants using data from the UK Biobank and found that the causal variant (single-nucleotide polymorphism rs8072917) was associated with muscle symptoms, cellulitis and abscess of face and neck, and cardiogenic shock. Our findings foster the genetic architecture of headache in individuals of East Asian ancestry. Our study can be replicated using genomic data linked to electronic health records from a variety of countries, therefore affecting a wide range of ethnicities globally. Our genome-phenome association study may facilitate the development of new genetic tests and novel drug mechanisms.

12,026 Taiwanese Han Chinese ancestry cases, 96,829 Taiwanese Han Chinese ancestry controls

Chapter III

Study Statistics

Key metrics and study information

108855
Total Participants
GWAS
Study Type
No
Replicated
East Asian
Ancestry
Taiwan
Recruitment Country
Chapter IV

Analysis

Comprehensive review of health and genetic findings

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