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GWAS Study

Phenotypic but not genetically predicted heart rate variability associated with all-cause mortality.

Tegegne BS, Said MA, Ani A et al.

37803156 PubMed ID
GWAS Study Type
46075 Participants
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Chapter I

Publication Details

Comprehensive information about this research publication

Authors

TB
Tegegne BS
SM
Said MA
AA
Ani A
VR
van Roon AM
SS
Shah S
DG
de Geus EJC
VD
van der Harst P
RH
Riese H
NI
Nolte IM
SH
Snieder H
Chapter II

Abstract

Summary of the research findings

Low heart rate variability (HRV) has been widely reported as a predictor for increased mortality. However, the molecular mechanisms are poorly understood. Therefore, this study aimed to identify novel genetic loci associated with HRV and assess the association of phenotypic HRV and genetically predicted HRV with mortality. In a GWAS of 46,075 European ancestry individuals from UK biobank, we identified 17 independent genome-wide significant genetic variants in 16 loci associated with HRV traits. Notably, eight of these loci (RNF220, GNB4, LINCR-002, KLHL3/HNRNPA0, CHRM2, KCNJ5, MED13L, and C160rf72) have not been reported previously. In a prospective phenotypic relationship between HRV and mortality during a median follow-up of seven years, individuals with lower HRV had higher risk of dying from any cause. Genetically predicted HRV, as determined by the genetic risk scores, was not associated with mortality. To the best of our knowledge, the findings provide novel biological insights into the mechanisms underlying HRV. These results also underline the role of the cardiac autonomic nervous system, as indexed by HRV, in predicting mortality.

46,075 European ancestry individuals

Chapter III

Study Statistics

Key metrics and study information

46075
Total Participants
GWAS
Study Type
No
Replicated
European
Ancestry
U.K.
Recruitment Country
Chapter IV

Analysis

Comprehensive review of health and genetic findings

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