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GWAS Study

A genome-wide association study of neutrophil count in individuals associated to an African continental ancestry group facilitates studies of malaria pathogenesis.

Constantinescu AE, Hughes DA, Bull CJ et al.

38491524 PubMed ID
GWAS Study Type
5976 Participants
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Chapter I

Publication Details

Comprehensive information about this research publication

Authors

CA
Constantinescu AE
HD
Hughes DA
BC
Bull CJ
FK
Fleming K
MR
Mitchell RE
ZJ
Zheng J
KS
Kar S
TN
Timpson NJ
AB
Amulic B
VE
Vincent EE
Chapter II

Abstract

Summary of the research findings

'Benign ethnic neutropenia' (BEN) is a heritable condition characterized by lower neutrophil counts, predominantly observed in individuals of African ancestry, and the genetic basis of BEN remains a subject of extensive research. In this study, we aimed to dissect the genetic architecture underlying neutrophil count variation through a linear-mixed model genome-wide association study (GWAS) in a population of African ancestry (N = 5976). Malaria caused by P. falciparum imposes a tremendous public health burden on people living in sub-Saharan Africa. Individuals living in malaria endemic regions often have a reduced circulating neutrophil count due to BEN, raising the possibility that reduced neutrophil counts modulate severity of malaria in susceptible populations. As a follow-up, we tested this hypothesis by conducting a Mendelian randomization (MR) analysis of neutrophil counts on severe malaria (MalariaGEN, N = 17,056).

5,976 African ancestry individuals

Chapter III

Study Statistics

Key metrics and study information

5976
Total Participants
GWAS
Study Type
No
Replicated
African unspecified
Ancestry
U.K.
Recruitment Country
Chapter IV

Analysis

Comprehensive review of health and genetic findings

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