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GWAS Study

Biological, clinical and population relevance of 95 loci for blood lipids.

Teslovich TM, Musunuru K, Smith AV et al.

20686565 PubMed ID
GWAS Study Type
96598 Participants
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Chapter I

Publication Details

Comprehensive information about this research publication

Authors

TT
Teslovich TM
MK
Musunuru K
SA
Smith AV
EA
Edmondson AC
SI
Stylianou IM
KM
Koseki M
PJ
Pirruccello JP
RS
Ripatti S
CD
Chasman DI
WC
Willer CJ
JC
Johansen CT
FS
Fouchier SW
IA
Isaacs A
PG
Peloso GM
BM
Barbalic M
RS
Ricketts SL
BJ
Bis JC
AY
Aulchenko YS
TG
Thorleifsson G
FM
Feitosa MF
CJ
Chambers J
OM
Orho-Melander M
MO
Melander O
JT
Johnson T
LX
Li X
GX
Guo X
LM
Li M
SC
Shin Cho Y
JG
Jin Go M
JK
Jin Kim Y
LJ
Lee JY
PT
Park T
KK
Kim K
SX
Sim X
TO
Twee-Hee Ong R
CD
Croteau-Chonka DC
LL
Lange LA
SJ
Smith JD
SK
Song K
HZ
Hua Zhao J
YX
Yuan X
LJ
Luan J
LC
Lamina C
ZA
Ziegler A
ZW
Zhang W
ZR
Zee RY
WA
Wright AF
WJ
Witteman JC
WJ
Wilson JF
WG
Willemsen G
WH
Wichmann HE
WJ
Whitfield JB
WD
Waterworth DM
WN
Wareham NJ
WG
Waeber G
VP
Vollenweider P
VB
Voight BF
VV
Vitart V
UA
Uitterlinden AG
UM
Uda M
TJ
Tuomilehto J
TJ
Thompson JR
TT
Tanaka T
SI
Surakka I
SH
Stringham HM
ST
Spector TD
SN
Soranzo N
SJ
Smit JH
SJ
Sinisalo J
SK
Silander K
SE
Sijbrands EJ
SA
Scuteri A
SJ
Scott J
SD
Schlessinger D
SS
Sanna S
SV
Salomaa V
SJ
Saharinen J
SC
Sabatti C
RA
Ruokonen A
RI
Rudan I
RL
Rose LM
RR
Roberts R
RM
Rieder M
PB
Psaty BM
PP
Pramstaller PP
PI
Pichler I
PM
Perola M
PB
Penninx BW
PN
Pedersen NL
PC
Pattaro C
PA
Parker AN
PG
Pare G
OB
Oostra BA
OC
O'Donnell CJ
NM
Nieminen MS
ND
Nickerson DA
MG
Montgomery GW
MT
Meitinger T
MR
McPherson R
MM
McCarthy MI
MW
McArdle W
MD
Masson D
MN
Martin NG
MF
Marroni F
MM
Mangino M
MP
Magnusson PK
LG
Lucas G
LR
Luben R
LR
Loos RJ
LM
Lokki ML
LG
Lettre G
LC
Langenberg C
LL
Launer LJ
LE
Lakatta EG
LR
Laaksonen R
KK
Kyvik KO
KF
Kronenberg F
KI
König IR
KK
Khaw KT
KJ
Kaprio J
KL
Kaplan LM
JA
Johansson A
JM
Jarvelin MR
JA
Janssens AC
IE
Ingelsson E
IW
Igl W
KH
Kees Hovingh G
HJ
Hottenga JJ
HA
Hofman A
HA
Hicks AA
HC
Hengstenberg C
HI
Heid IM
HC
Hayward C
HA
Havulinna AS
HN
Hastie ND
HT
Harris TB
HT
Haritunians T
HA
Hall AS
GU
Gyllensten U
GC
Guiducci C
GL
Groop LC
GE
Gonzalez E
GC
Gieger C
FN
Freimer NB
FL
Ferrucci L
EJ
Erdmann J
EP
Elliott P
EK
Ejebe KG
DA
Döring A
DA
Dominiczak AF
DS
Demissie S
DP
Deloukas P
DG
de Geus EJ
DF
de Faire U
CG
Crawford G
CF
Collins FS
CY
Chen YD
CM
Caulfield MJ
CH
Campbell H
BN
Burtt NP
BL
Bonnycastle LL
BD
Boomsma DI
BS
Boekholdt SM
BR
Bergman RN
BI
Barroso I
BS
Bandinelli S
BC
Ballantyne CM
AT
Assimes TL
QT
Quertermous T
AD
Altshuler D
SM
Seielstad M
WT
Wong TY
TE
Tai ES
FA
Feranil AB
KC
Kuzawa CW
AL
Adair LS
TH
Taylor HA
BI
Borecki IB
GS
Gabriel SB
WJ
Wilson JG
HH
Holm H
TU
Thorsteinsdottir U
GV
Gudnason V
KR
Krauss RM
MK
Mohlke KL
OJ
Ordovas JM
MP
Munroe PB
KJ
Kooner JS
TA
Tall AR
HR
Hegele RA
KJ
Kastelein JJ
SE
Schadt EE
RJ
Rotter JI
BE
Boerwinkle E
SD
Strachan DP
MV
Mooser V
SK
Stefansson K
RM
Reilly MP
SN
Samani NJ
SH
Schunkert H
CL
Cupples LA
SM
Sandhu MS
RP
Ridker PM
RD
Rader DJ
VD
van Duijn CM
PL
Peltonen L
AG
Abecasis GR
BM
Boehnke M
KS
Kathiresan S
Chapter II

Abstract

Summary of the research findings

Plasma concentrations of total cholesterol, low-density lipoprotein cholesterol, high-density lipoprotein cholesterol and triglycerides are among the most important risk factors for coronary artery disease (CAD) and are targets for therapeutic intervention. We screened the genome for common variants associated with plasma lipids in >100,000 individuals of European ancestry. Here we report 95 significantly associated loci (P < 5 x 10(-8)), with 59 showing genome-wide significant association with lipid traits for the first time. The newly reported associations include single nucleotide polymorphisms (SNPs) near known lipid regulators (for example, CYP7A1, NPC1L1 and SCARB1) as well as in scores of loci not previously implicated in lipoprotein metabolism. The 95 loci contribute not only to normal variation in lipid traits but also to extreme lipid phenotypes and have an impact on lipid traits in three non-European populations (East Asians, South Asians and African Americans). Our results identify several novel loci associated with plasma lipids that are also associated with CAD. Finally, we validated three of the novel genes-GALNT2, PPP1R3B and TTC39B-with experiments in mouse models. Taken together, our findings provide the foundation to develop a broader biological understanding of lipoprotein metabolism and to identify new therapeutic opportunities for the prevention of CAD.

96,598 European ancestry individuals

Chapter III

Study Statistics

Key metrics and study information

96598
Total Participants
GWAS
Study Type
No
Replicated
European
Ancestry
Finland, Sweden, U.S., Australia, Iceland, Italy, Netherlands, Germany, U.K., Croatia, Switzerland, Austria, France, Denmark
Recruitment Country
Chapter IV

Analysis

Comprehensive review of health and genetic findings

Important Disclaimer: This review has been performed semi-automatically and is provided for informational purposes only. While we strive for accuracy, this analysis may contain errors, omissions, or misinterpretations of the original research. DNA Genics disclaims all liability for any inaccuracies, errors, or consequences arising from the use of this information. Users should independently verify all information and consult original research publications before making any decisions based on this content. This analysis is not intended as a substitute for professional scientific review or medical advice.

Analysis In Progress

Our analysis of this publication is currently being prepared. Please check back soon for comprehensive insights into the health and genetic findings discussed in this research.