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GWAS Study

Genome-wide association study of a nicotine metabolism biomarker in African American smokers: impact of chromosome 19 genetic influences.

Chenoweth MJ, Ware JJ, Zhu AZX et al.

28921760 PubMed ID
GWAS Study Type
1434 Participants
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Chapter I

Publication Details

Comprehensive information about this research publication

Authors

CM
Chenoweth MJ
WJ
Ware JJ
ZA
Zhu AZX
CC
Cole CB
CL
Cox LS
NN
Nollen N
AJ
Ahluwalia JS
BN
Benowitz NL
SR
Schnoll RA
HL
Hawk LW
CP
Cinciripini PM
GT
George TP
LC
Lerman C
KJ
Knight J
TR
Tyndale RF
Chapter II

Abstract

Summary of the research findings

Background and aims: The activity of CYP2A6, the major nicotine-inactivating enzyme, is measurable in smokers using the nicotine metabolite ratio (NMR; 3'hydroxycotinine/cotinine). Due to its role in nicotine clearance, the NMR is associated with smoking behaviours and response to pharmacotherapies. The NMR is highly heritable (~80%), and on average lower in African Americans (AA) versus whites. We previously identified several reduce and loss-of-function CYP2A6 variants common in individuals of African descent. Our current aim was to identify novel genetic influences on the NMR in AA smokers using genome-wide approaches.

954 African American individuals

Chapter III

Study Statistics

Key metrics and study information

1434
Total Participants
GWAS
Study Type
Yes
Replicated
480 African American individuals
Replication Participants
African American or Afro-Caribbean
Ancestry
U.S., Canada
Recruitment Country
Chapter IV

Analysis

Comprehensive review of health and genetic findings

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