Menu
GWAS Study

Novel loci and biomedical consequences of iron homoeostasis variation.

Allara E, Bell S, Smith R et al.

39643614 PubMed ID
GWAS Study Type
91675 Participants
Scroll to explore
Chapter I

Publication Details

Comprehensive information about this research publication

Authors

AE
Allara E
BS
Bell S
SR
Smith R
KS
Keene SJ
GD
Gill D
GL
Gaziano L
MG
Morselli Gysi D
WF
Wang F
TV
Tragante V
MA
Mason A
KS
Karthikeyan S
LR
Lumbers RT
BE
Bonglack E
OW
Ouwehand W
RD
Roberts DJ
DJ
Dowsett J
OS
Ostrowski SR
LM
Larsen MH
UH
Ullum H
PO
Pedersen OB
BS
Brunak S
BK
Banasik K
EC
Erikstrup C
MJ
Mitchell J
FC
Fuchsberger C
PC
Pattaro C
PP
Pramstaller PP
GD
Girelli D
AM
Arvas M
TJ
Toivonen J
MS
Molnos S
PA
Peters A
PO
Polasek O
RI
Rudan I
HC
Hayward C
MC
McDonnell C
PN
Pirastu N
WJ
Wilson JF
VD
van den Hurk K
QF
Quee F
FL
Ferrucci L
BS
Bandinelli S
TT
Tanaka T
GG
Girotto G
CM
Concas MP
PA
Pecori A
VN
Verweij N
VD
van der Harst P
VD
van de Vegte YJ
KL
Kiemeney LA
SF
Sweep FC
GT
Galesloot TE
SP
Sulem P
GD
Gudbjartsson D
FE
Ferkingstad E
DL
Djousse L
CK
Cho K
IM
Inouye M
BS
Burgess S
BB
Benyamin B
OK
Oexle K
SD
Swinkels D
SK
Stefansson K
MM
Magnusson M
GA
Ganna A
GM
Gaziano M
IK
Ivey K
DJ
Danesh J
PA
Pereira A
WA
Wood AM
BA
Butterworth AS
DA
Di Angelantonio E
Chapter II

Abstract

Summary of the research findings

Iron homoeostasis is tightly regulated, with hepcidin and soluble transferrin receptor (sTfR) playing significant roles. However, the genetic determinants of these traits and the biomedical consequences of iron homoeostasis variation are unclear. In a meta-analysis of 12 cohorts involving 91,675 participants, we found 43 genomic loci associated with either hepcidin or sTfR concentration, of which 15 previously unreported. Mapping to putative genes indicated involvement in iron-trait expression, erythropoiesis, immune response and cellular trafficking. Mendelian randomisation of 292 disease outcomes in 1,492,717 participants revealed associations of iron-related loci and iron status with selected health outcomes across multiple domains. These associations were largely driven by HFE, which was associated with the largest iron variation. Our findings enhance understanding of iron homoeostasis and its biomedical consequences, suggesting that lifelong exposure to higher iron levels is likely associated with lower risk of anaemia-related disorders and higher risk of genitourinary, musculoskeletal, infectious and neoplastic diseases.

91,675 European ancestry individuals

Chapter III

Study Statistics

Key metrics and study information

91675
Total Participants
GWAS
Study Type
No
Replicated
European
Ancestry
Chapter IV

Analysis

Comprehensive review of health and genetic findings

Important Disclaimer: This review has been performed semi-automatically and is provided for informational purposes only. While we strive for accuracy, this analysis may contain errors, omissions, or misinterpretations of the original research. DNA Genics disclaims all liability for any inaccuracies, errors, or consequences arising from the use of this information. Users should independently verify all information and consult original research publications before making any decisions based on this content. This analysis is not intended as a substitute for professional scientific review or medical advice.

Analysis In Progress

Our analysis of this publication is currently being prepared. Please check back soon for comprehensive insights into the health and genetic findings discussed in this research.