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GWAS Study

Genomics of chronic dry cough unravels neurological pathways.

Coley K, John C, Ghouse J et al.

40675770 PubMed ID
GWAS Study Type
180380 Participants
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Chapter I

Publication Details

Comprehensive information about this research publication

Authors

CK
Coley K
JC
John C
GJ
Ghouse J
SD
Shepherd DJ
SN
Shrine N
IA
Izquierdo AG
KS
Kanoni S
ME
Magavern EF
PR
Packer R
ML
McGarvey L
SJ
Smith JA
BH
Bundgaard H
OS
Ostrowski SR
EC
Erikstrup C
PO
Pedersen OBV
VH
van Heel DA
HW
Hennah W
MM
Marttila M
FR
Free RC
HE
Hollox EJ
WL
Wain LV
TM
Tobin MD
BC
Batini C
Chapter II

Abstract

Summary of the research findings

Chronic dry cough is a symptom of common lung conditions, can occur as a side-effect of angiotensin-converting enzyme inhibitors (ACEis), or may be unexplained. Despite the substantial health burden presented by chronic dry cough, its biological mechanisms remain unclear. We hypothesised shared genetic architecture between chronic dry cough and ACEi-induced cough and aimed to identify causal genes underlying both phenotypes.

226 African ancestry cases, 1,061 African ancestry controls, 27,772 European ancestry cases, 141,390 European ancestry controls, 32 East Asian ancestry cases, 116 East Asian ancestry controls, 1,057 South Asian ancestry cases, 8,532 South Asian ancestry controls, 71 cases, 123 controls

Chapter III

Study Statistics

Key metrics and study information

180380
Total Participants
GWAS
Study Type
No
Replicated
African unspecified, European, East Asian, South Asian
Ancestry
U.K., U.S., Denmark
Recruitment Country
Chapter IV

Analysis

Comprehensive review of health and genetic findings

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Analysis In Progress

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